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Insight into the circadian system of the spontaneously hypertensive rat (SHR) during ontogenesis and in adulthood

Project goals

The mammalian circadian system exhibits a temporal control over physiological functions, including energy metabolism. Perturbation of the circadian organization may affect the energy metabolism and, vice versa, alterations in the metabolism may feed backon the circadian system. The spontaneously hypertensive rat (SHR) shows many traits associated with metabolic syndrome and its inherent metabolic state is different in comparison with control animals. The aim of the proposal is to elucidate whether andhow the SHR circadian system differs from that in control rats, both during ontogenesis and in adulthood. The circadian behavioral phenotype and molecular mechanisms of the core clockwork in the circadian clock in the brain and in the peripheral clocks related to metabolism will be studied. Moreover, development of the circadian system and its maternal entrainment in the SHR will be determined. The results will provide for the first time an insight into molecular mechanisms of the SHR circadian system and thus into its function in conditions of genetically altered metabolic state.

Keywords

circadian system ontogenesis SHR

Public support

  • Provider

    Czech Science Foundation

  • Programme

    Standard projects

  • Call for proposals

    Standardní projekty 14 (SGA02011GA-ST)

  • Main participants

  • Contest type

    VS - Public tender

  • Contract ID

    P303-11-0668

Alternative language

  • Project name in Czech

    Cirkadiánní systém potkana kmene SHR během ontogeneze a v dospělosti

  • Annotation in Czech

    Cirkadiánní systém savců řídí časově řadu základních fyziologických funkcí včetně metabolismu. Porucha časové organizace má významný dopad na energetický metabolismus a naopak, změny v metabolismu mohou zpětně ovlivnit cirkadiánní systém. Spontánně hypertenzivní potkan (SHR) vykazuje mnoho znaků spojovaných s metabolickým syndromem a jeho vrozený metabolický stav se liší od kontrolních zvířat. Cílem studie je zjistit, zda a jak je cirkadiánní systém SHR změněn během ontogeneze a v dospělosti ve srovnánís kontrolními zvířaty. Bude zkoumán cirkadiánní behaviorální fenotyp SHR a molekulární mechanismy chodu jeho cirkadiánních hodin v suprachiasmatických jádrech a v periferních tkáních související s metabolismem. Kromě toho bude studován vývoj cirkadiánního systému SHR a jeho synchronizace matkou. Výsledky poskytnou primární informace o molekulárních mechanismech cirkadiánního systému u SHR a pomohou tak zjistit jak je tento systém ovlivňován genetickou změnou metabolismu.

Scientific branches

  • R&D category

    ZV - Basic research

  • CEP classification - main branch

    ED - Physiology

  • CEP - secondary branch

    FH - Neurology, neuro-surgery, nuero-sciences

  • CEP - another secondary branch

  • 30103 - Neurosciences (including psychophysiology)
    30105 - Physiology (including cytology)
    30210 - Clinical neurology

Completed project evaluation

  • Provider evaluation

    V - Vynikající výsledky projektu (s mezinárodním významem atd.)

  • Project results evaluation

    Project results demonstrate that circadian system of spontaneously hypertensive rats exhibits significant aberrancies compared with the healthy control rats. The central circadian clock in SHR runs faster, misaligned with external light/dark cycle and drives dampened rhythms in behavior, exhibiting early chronotype-like characteristics. Results were published in 6 publication of the high quality.

Solution timeline

  • Realization period - beginning

    Jan 1, 2011

  • Realization period - end

    Dec 31, 2013

  • Project status

    U - Finished project

  • Latest support payment

    Jun 6, 2013

Data delivery to CEP

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

  • Data delivery code

    CEP14-GA0-GA-U/01:1

  • Data delivery date

    Jul 1, 2014

Finance

  • Total approved costs

    4,920 thou. CZK

  • Public financial support

    4,920 thou. CZK

  • Other public sources

    0 thou. CZK

  • Non public and foreign sources

    0 thou. CZK

Basic information

Recognised costs

4 920 CZK thou.

Public support

4 920 CZK thou.

100%


Provider

Czech Science Foundation

CEP

ED - Physiology

Solution period

01. 01. 2011 - 31. 12. 2013