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The role of PAR-2 in neurodegenerative diseases

Project goals

Proteinase-activated receptors are essentially ubiquitous receptors activated by certain serine proteinases. This is a family of G-protein coupled receptors activated by subsequent cleavage at the specific site of the NH2 terminal and presentation of the new terminal, which behaves as "a tethered ligand". Neurodegenerative diseases are very serious confirmed diseases which incidence is sharply increasing in relation to the aging of the population. Based on the literature and according to our previous finding we assume that serum proteinases play an important role in the pathophysiology of degenerative diseases of CNS via activation of PAR´s. We further assume that these processes are closely linked to the blood-brain barrier damage by endogenous or exogenous factors. To observe the role of PAR in degenerative processes of CNS the PAR-2 changes of expressions will be followed and compared to other neurodegenerative markers in animal models. The obtained results will be compared to the observation of the changes of expression of PAR´s in the model of human samples of neurodegenerative disorders. The human samples will be obtained from the collection of neuropathological material of autoptic CNS tissues affected by different neurodegenerative diseases.

Keywords

PAR-2neurodegeneracetrypsin

Public support

  • Provider

    Czech Science Foundation

  • Programme

    Post-graduate (doctorate) grants

  • Call for proposals

    Postdoktorandské granty 9 (SGA02009GA1PD)

  • Main participants

  • Contest type

    VS - Public tender

  • Contract ID

    309/09/P204

Alternative language

  • Project name in Czech

    Úloha PAR-2 v neurodegenerativních onemocněních

  • Annotation in Czech

    Cílem předkládaného projektu je zjistit zda a jakým způsobem se proteinázy trypsin a trombin a jejich agonistické molekuly podílí na patogenezi neurodegenerativních onemocnění prostřednictvím aktivace PAR´s.

Scientific branches

  • R&D category

    ZV - Basic research

  • CEP classification - main branch

    FH - Neurology, neuro-surgery, nuero-sciences

  • CEP - secondary branch

    EB - Genetics and molecular biology

  • CEP - another secondary branch

    EA - Morphology and cytology

  • 10601 - Cell biology
    10602 - Biology (theoretical, mathematical, thermal, cryobiology, biological rhythm), Evolutionary biology
    10603 - Genetics and heredity (medical genetics to be 3)
    10604 - Reproductive biology (medical aspects to be 3)
    10605 - Developmental biology
    10608 - Biochemistry and molecular biology
    10609 - Biochemical research methods
    30101 - Human genetics
    30103 - Neurosciences (including psychophysiology)
    30210 - Clinical neurology

Completed project evaluation

  • Provider evaluation

    V - Vynikající výsledky projektu (s mezinárodním významem atd.)

  • Project results evaluation

    The project was excellent, both in terms of expertise and in terms of disbursement of funds.

Solution timeline

  • Realization period - beginning

    Jan 1, 2009

  • Realization period - end

    Dec 31, 2011

  • Project status

    U - Finished project

  • Latest support payment

    Apr 16, 2011

Data delivery to CEP

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

  • Data delivery code

    CEP12-GA0-GP-U/03:3

  • Data delivery date

    May 2, 2016

Finance

  • Total approved costs

    1,797 thou. CZK

  • Public financial support

    1,797 thou. CZK

  • Other public sources

    0 thou. CZK

  • Non public and foreign sources

    0 thou. CZK

Basic information

Recognised costs

1 797 CZK thou.

Public support

1 797 CZK thou.

100%


Provider

Czech Science Foundation

CEP

FH - Neurology, neuro-surgery, nuero-sciences

Solution period

01. 01. 2009 - 31. 12. 2011