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Rosuvastatin can block pro-inflammatory actions of transgenic human C-reactive protein without reducing its circulating levels

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023001%3A_____%2F14%3A00058924" target="_blank" >RIV/00023001:_____/14:00058924 - isvavai.cz</a>

  • Alternative codes found

    RIV/67985823:_____/14:00427797

  • Result on the web

    <a href="http://onlinelibrary.wiley.com/doi/10.1111/1755-5922.12061/abstract" target="_blank" >http://onlinelibrary.wiley.com/doi/10.1111/1755-5922.12061/abstract</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1111/1755-5922.12061" target="_blank" >10.1111/1755-5922.12061</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Rosuvastatin can block pro-inflammatory actions of transgenic human C-reactive protein without reducing its circulating levels

  • Original language description

    AimsStatins have antiinflammatory effects and are known to decrease risk of cardiovascular events and to reduce serum levels of C-reactive protein (CRP), a widely studied biomarker and potential mediator of inflammation and heart disease. However, it isunclear whether statins can block pro-inflammatory effects of human CRP independent of their ability to reduce serum levels of human CRP. Here, we investigated whether rosuvastatin could block pro-inflammatory effects of human CRP without reducing circulating levels of human CRP. Methods and ResultsWe studied the antiinflammatory effects of rosuvastatin in spontaneously hypertensive rats (SHR) transgenically expressing human CRP (CRP-transgenic SHR) and in nontransgenic SHR lacking human CRP (nontransgenic SHR). The CRP-transgenic SHR is characterized by increased serum levels of human CRP and inflammation. In the CRP-transgenic strain, we found that rosuvastatin treatment decreased circulating levels of inflammatory response markers IL

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    FB - Endocrinology, diabetology, metabolism, nutrition

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2014

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Cardiovascular therapeutics

  • ISSN

    1755-5914

  • e-ISSN

  • Volume of the periodical

    32

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    7

  • Pages from-to

    59-65

  • UT code for WoS article

    000332766000006

  • EID of the result in the Scopus database