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Coronary artery disease is associated with an increased amount of T lymphocytes in human epicardial adipose tissue

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023001%3A_____%2F19%3A00077674" target="_blank" >RIV/00023001:_____/19:00077674 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11110/19:10392504 RIV/00064165:_____/19:10392504

  • Result on the web

    <a href="https://www.hindawi.com/journals/mi/2019/4075086/" target="_blank" >https://www.hindawi.com/journals/mi/2019/4075086/</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1155/2019/4075086" target="_blank" >10.1155/2019/4075086</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Coronary artery disease is associated with an increased amount of T lymphocytes in human epicardial adipose tissue

  • Original language description

    Immunocompetent cells including lymphocytes play a key role in the development of adipose tissue inflammation and obesity-related cardiovascular complications. The aim of the study was to explore the relationship between epicardial adipose tissue lymphocytes and coronary artery disease (CAD). To this end, we studied the content and phenotype of lymphocytes in peripheral blood, subcutaneous adipose tissue (SAT), and epicardial adipose tissue (EAT) in subjects with and without CAD undergoing elective cardiac surgery. Eleven subjects without CAD (non-CAD group) and 22 age-, BMI-, and HbA(1C)-matched individuals with CAD were included into the study. Blood, SAT, and EAT samples were obtained at the beginning of surgery. Lymphocyte populations were quantified as % of CD45+ cells using flow cytometry. Subjects with CAD had a higher total lymphocyte amount in EAT compared with SAT (32.24 +/- 7.45 vs. 11.22 +/- 1.34%, p=0.025) with a similar trend observed in non-CAD subjects (29.68 +/- 7.61 vs. 10.13 +/- 2.01%, p=0.067). T (CD3+) cells were increased (75.33 +/- 2.18 vs. 65.24 +/- 4.49%, p=0.032) and CD3- cells decreased (21.17 +/- 2.26 vs. 31.64 +/- 4.40%, p=0.028) in EAT of CAD relative to the non-CAD group. In both groups, EAT showed an elevated percentage of B cells (5.22 +/- 2.43 vs. 0.96 +/- 0.21%, p=0.039 for CAD and 12.49 +/- 5.83 vs. 1.16 +/- 0.19%, p=0.016 for non-CAD) and reduced natural killer (NK) cells (5.96 +/- 1.32 vs. 13.22 +/- 2.10%, p=0.012 for CAD and 5.32 +/- 1.97 vs. 13.81 +/- 2.72%, p=0.022 for non-CAD) relative to SAT. In conclusion, epicardial adipose tissue in subjects with CAD shows an increased amount of T lymphocytes relative to non-CAD individuals as well as a higher number of total and B lymphocytes and reduced NK cells as compared with corresponding SAT. These changes could contribute to the development of local inflammation and coronary atherosclerosis.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30202 - Endocrinology and metabolism (including diabetes, hormones)

Result continuities

  • Project

    <a href="/en/project/NV15-26854A" target="_blank" >NV15-26854A: The role of epicardial adipose tissue in heart disease: novel mechanisms of action and its therapeutic modulation</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2019

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Mediators of inflammation [online]

  • ISSN

    0962-9351

  • e-ISSN

  • Volume of the periodical

    2019

  • Issue of the periodical within the volume

    February 2019

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    9

  • Pages from-to

    "art. no. 4075086"

  • UT code for WoS article

    000459077000001

  • EID of the result in the Scopus database

    2-s2.0-85063274859