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Cerebral oxygen saturation and autoregulation during hypotension in extremely preterm infants

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023698%3A_____%2F21%3AN0000018" target="_blank" >RIV/00023698:_____/21:N0000018 - isvavai.cz</a>

  • Alternative codes found

    RIV/00843989:_____/21:E0109199 RIV/00216208:11120/21:43921423 RIV/70883521:28150/21:63527778

  • Result on the web

    <a href="https://doi.org/10.1038/s41390-021-01483-w" target="_blank" >https://doi.org/10.1038/s41390-021-01483-w</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/s41390-021-01483-w" target="_blank" >10.1038/s41390-021-01483-w</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Cerebral oxygen saturation and autoregulation during hypotension in extremely preterm infants

  • Original language description

    Background: The impact of the permissive hypotension approach in clinically well infants on regional cerebral oxygen saturation (rScO2) and autoregulatory capacity (CAR) remains unknown. Methods: Prospective cohort study of blinded rScO2 measurements within a randomized controlled trial of management of hypotension (HIP trial) in extremely preterm infants. rScO2, mean arterial blood pressure, duration of cerebral hypoxia, and transfer function (TF) gain inversely proportional to CAR, were compared between hypotensive infants randomized to receive dopamine or placebo and between hypotensive and non-hypotensive infants, and related to early intraventricular hemorrhage or death. Results: In 89 potentially eligible HIP trial patients with rScO2 measurements, the duration of cerebral hypoxia was significantly higher in 36 hypotensive compared to 53 non-hypotensive infants. In 29/36 hypotensive infants (mean GA 25 weeks, 69% males) receiving the study drug, no significant difference in rScO2 was observed after dopamine (n = 13) compared to placebo (n = 16). Duration of cerebral hypoxia was associated with early intraventricular hemorrhage or death. Calculated TF gain (n = 49/89) was significantly higher reflecting decreased CAR in 16 hypotensive compared to 33 non-hypotensive infants. Conclusions: Dopamine had no effect on rScO2 compared to placebo in hypotensive infants. Hypotension and cerebral hypoxia are associated with early intraventricular hemorrhage or death. Impact: Treatment of hypotension with dopamine in extremely preterm infants increases mean arterial blood pressure, but does not improve cerebral oxygenation.Hypotensive extremely preterm infants have increased duration of cerebral hypoxia and reduced cerebral autoregulatory capacity compared to non-hypotensive infants.Duration of cerebral hypoxia and hypotension are associated with early intraventricular hemorrhage or death in extremely preterm infants.Since systematic treatment of hypotension may not be associated with better outcomes, the diagnosis of cerebral hypoxia in hypotensive extremely preterm infants might guide treatment. © 2021, The Author(s), under exclusive licence to the International Pediatric Research Foundation, Inc. Reaxys Chemistry database informationLearn about Reaxys chemistry database information

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30209 - Paediatrics

Result continuities

  • Project

  • Continuities

    R - Projekt Ramcoveho programu EK

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Pediatric Research

  • ISSN

    0031-3998

  • e-ISSN

    1530-0447

  • Volume of the periodical

    90

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    8

  • Pages from-to

    373-380

  • UT code for WoS article

    000641661500006

  • EID of the result in the Scopus database

    2-s2.0-85105111800