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Genetic background of uric acid metabolism in patient with severe chronic tophaceous gout

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023728%3A_____%2F16%3AN0000034" target="_blank" >RIV/00023728:_____/16:N0000034 - isvavai.cz</a>

  • Result on the web

    <a href="https://doi.org/10.1016/j.cca.2016.06.007" target="_blank" >https://doi.org/10.1016/j.cca.2016.06.007</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.cca.2016.06.007" target="_blank" >10.1016/j.cca.2016.06.007</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Genetic background of uric acid metabolism in patient with severe chronic tophaceous gout

  • Original language description

    Hyperuricemia depends on the balance of endogenous production and renal excretion of uric acid. Transporters for urate are located in the proximal tubule where uric acid is secreted and extensively reabsorbed: secretion is principally ensured by the highly variable ABCG2 gene. Enzyme hypoxanthine–guanine phosphoribosyltransferase (HPRT) plays a central role in purine metabolism and its deficiency is an X-linked inherited metabolic disorder associated with clinical manifestations of purine overproduction. Here we report the case of a middle-aged man with severe chronic tophaceous gout with a poor response to allopurinol and requiring repeated surgical intervention. We identified the causal mutations in the HPRT1 gene, variant c.481G > T (p.A161S), and in the crucial urate transporter ABCG2, a heterozygous variant c.421C > A (p.Q141K). This case shows the value of an analysis of the genetic background of serum uric acid.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30226 - Rheumatology

Result continuities

  • Project

    <a href="/en/project/NV15-26693A" target="_blank" >NV15-26693A: Function study of allelic variants of urate transporters in primary hyperuricemia and gout</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2016

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Clinica Chimica acta 2016, 460, s. 46-49

  • ISSN

    0009-8981

  • e-ISSN

    1873-3492

  • Volume of the periodical

    460

  • Issue of the periodical within the volume

    2016

  • Country of publishing house

    NL - THE KINGDOM OF THE NETHERLANDS

  • Number of pages

    4

  • Pages from-to

    46-49

  • UT code for WoS article

    000382352600008

  • EID of the result in the Scopus database

    2-s2.0-84975801808