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Transcriptional and Epigenetic Mechanisms Underlying Enhanced in Vitro Adipocyte Differentiation by the Brominated Flame Retardant BDE-47

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00027162%3A_____%2F14%3A%230001184" target="_blank" >RIV/00027162:_____/14:#0001184 - isvavai.cz</a>

  • Result on the web

    <a href="http://dx.doi.org/10.1021/es405524b" target="_blank" >http://dx.doi.org/10.1021/es405524b</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1021/es405524b" target="_blank" >10.1021/es405524b</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Transcriptional and Epigenetic Mechanisms Underlying Enhanced in Vitro Adipocyte Differentiation by the Brominated Flame Retardant BDE-47

  • Original language description

    Recent studies suggest that exposure to endocrine-disrupting compounds (EDCs) may play a role in the development of obesity. EDCs such as the flame retardant 2,2',4,4'-tetrabrominated diphenyl ether (BDE-47) have been shown to enhance adipocyte differentiation in the murine 3T3-L1 model. The mechanisms by which EDCs direct preadipocytes to form adipocytes are poorly understood. Here, we examined transcriptional and epigenetic mechanisms underlying the induction of in vitro adipocyte differentiation by BDE-47. Quantitative high content microscopy revealed concentration-dependent enhanced adipocyte differentiation following exposure to BDE-47 or the antidiabetic drug troglitazone (TROG). BDE-47 modestly activated the key adipogenic transcription factor peroxisome proliferator-activated receptor gamma (PPAR gamma) in COS7 cells, transiently transfected with a GAL4 reporter construct. Increased gene expression was observed for Ppar gamma 2, leptin (Lep), and glucose-6-phophatase catalytic

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    DN - Environmental impact on health

  • OECD FORD branch

Result continuities

  • Project

  • Continuities

    N - Vyzkumna aktivita podporovana z neverejnych zdroju

Others

  • Publication year

    2014

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Environmental Science & Technology

  • ISSN

    0013-936X

  • e-ISSN

  • Volume of the periodical

    48

  • Issue of the periodical within the volume

    7

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    10

  • Pages from-to

    4110-4119

  • UT code for WoS article

    000333776100062

  • EID of the result in the Scopus database