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A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00064203%3A_____%2F22%3A10433197" target="_blank" >RIV/00064203:_____/22:10433197 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11130/22:10433197 RIV/00216224:14740/22:00128828

  • Result on the web

    <a href="https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=iL0TRFRqJn" target="_blank" >https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=iL0TRFRqJn</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/s41590-021-01030-z" target="_blank" >10.1038/s41590-021-01030-z</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    A global effort to dissect the human genetic basis of resistance to SARS-CoV-2 infection

  • Original language description

    SARS-CoV-2 infections display tremendous interindividual variability, ranging from asymptomatic infections to life-threatening disease. Inborn errors of, and autoantibodies directed against, type I interferons (IFNs) account for about 20% of critical COVID-19 cases among SARS-CoV-2-infected individuals. By contrast, the genetic and immunological determinants of resistance to infection per se remain unknown. Following the discovery that autosomal recessive deficiency in the DARC chemokine receptor confers resistance to Plasmodium vivax, autosomal recessive deficiencies of chemokine receptor 5 (CCR5) and the enzyme FUT2 were shown to underlie resistance to HIV-1 and noroviruses, respectively. Along the same lines, we propose a strategy for identifying, recruiting, and genetically analyzing individuals who are naturally resistant to SARS-CoV-2 infection.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30102 - Immunology

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2022

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Nature Immunology

  • ISSN

    1529-2908

  • e-ISSN

    1529-2916

  • Volume of the periodical

    23

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    6

  • Pages from-to

    159-164

  • UT code for WoS article

    000708815900001

  • EID of the result in the Scopus database

    2-s2.0-85117437869