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BRCA Gene Mutations and Prostate Cancer

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00098892%3A_____%2F23%3A10158493" target="_blank" >RIV/00098892:_____/23:10158493 - isvavai.cz</a>

  • Alternative codes found

    RIV/61989592:15110/23:73616229

  • Result on the web

    <a href="https://www.intechopen.com/chapters/85044" target="_blank" >https://www.intechopen.com/chapters/85044</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.5772/intechopen.108792" target="_blank" >10.5772/intechopen.108792</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    BRCA Gene Mutations and Prostate Cancer

  • Original language description

    Prostate cancer remains the second most common cancer in men, with diverse courses from indolent cases to aggressive diseases. Among the key factors implicated in its pathogenesis are genomic alterations such as the TMPRSS2-ERG and related fusion oncogenes, loss of tumor suppressor PTEN, p53 or NKX3.1, inflammation, enhanced DNA damage, and chromosomal instability. Men with prostate cancer who carry BRCA1/2 mutations are at more risk of worse disease and poor prognosis. Cancer cells with mutant BRCA1 or BRCA2 repair genes with defects in homologous recombination are vulnerable to PARP inhibitors that target the genetic phenomenon known as synthetic lethality to exploit faulty DNA repair mechanisms. With relevance to prostate cancer, other features of cancer cells may also sensitize to PARP inhibitors, including aberrant transcription due to the androgen-driven fusion oncogene TMPRSS2-ERG or PTEN loss. Several models of synthetic lethality and potential biomarkers suggested up to date are also discussed. The chapter also highlights the importance of genetic screening of men with BRCA and shows diagnostic utility of plasma-derived circulating tumor DNA.

  • Czech name

  • Czech description

Classification

  • Type

    C - Chapter in a specialist book

  • CEP classification

  • OECD FORD branch

    30109 - Pathology

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2023

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Book/collection name

    BRCA1 and BRCA2 Mutations : Diagnostic and Therapeutic Implications

  • ISBN

    978-1-80356-806-5

  • Number of pages of the result

    9

  • Pages from-to

    nestrankovano

  • Number of pages of the book

    134

  • Publisher name

    IntechOpen Limited

  • Place of publication

    London

  • UT code for WoS chapter