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Uric acid modulates vascular endothelial function through the down regulation of nitric oxide production

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00159816%3A_____%2F13%3A00060710" target="_blank" >RIV/00159816:_____/13:00060710 - isvavai.cz</a>

  • Alternative codes found

    RIV/68081707:_____/13:00392583

  • Result on the web

    <a href="http://dx.doi.org/10.3109/10715762.2012.747677" target="_blank" >http://dx.doi.org/10.3109/10715762.2012.747677</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3109/10715762.2012.747677" target="_blank" >10.3109/10715762.2012.747677</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Uric acid modulates vascular endothelial function through the down regulation of nitric oxide production

  • Original language description

    Endothelial dysfunction characterized by decreased nitric oxide (NO) bioavailability is the first stage of coronary artery disease. It is known that one of the factors associated with an increased risk of coronary artery disease is a high plasma level ofuric acid. However, causative associations between hyperuricaemia and cardiovascular risk have not been definitely proved. In this work, we tested the effect of uric acid on endothelial NO bioavailability. Electrochemical measurement of NO production inacetylcholine-stimulated human umbilical endothelial cells (HUVECs) revealed that uric acid markedly decreases NO release. This finding was confirmed by organ bath experiments on mouse aortic segments. Uric acid dose-dependently reduced endothelium-dependent vasorelaxation. To reveal the mechanism of decreasing NO bioavailability we tested the effect of uric acid on reactive oxygen species production by HUVECs, on arginase activity, and on acetylcholine-induced endothelial NO synthase p

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    CE - Biochemistry

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Free radical research

  • ISSN

    1071-5762

  • e-ISSN

  • Volume of the periodical

    47

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    7

  • Pages from-to

    82-88

  • UT code for WoS article

    000313594600003

  • EID of the result in the Scopus database