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p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00209805%3A_____%2F13%3A%230000432" target="_blank" >RIV/00209805:_____/13:#0000432 - isvavai.cz</a>

  • Result on the web

    <a href="http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3859419/pdf/JCI70355.pdf" target="_blank" >http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3859419/pdf/JCI70355.pdf</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1172/JCI70355" target="_blank" >10.1172/JCI70355</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytes

  • Original language description

    Cellular senescence contributes to aging and decline in tissue function. p53 isoform switching regulates replicative senescence in cultured fibroblasts and is associated with tumor progression. Here, we found that the endogenous p53 isoforms ?133p53 andp53? are physiological regulators of proliferation and senescence in human T lymphocytes in vivo. Peripheral blood CD8+ T lymphocytes collected from healthy donors displayed an age-dependent accumulation of senescent cells (CD28?CD57+) with decreased ?133p53 and increased p53? expression. Human lung tumor-associated CD8+ T lymphocytes also harbored senescent cells. Cultured CD8+ blood T lymphocytes underwent replicative senescence that was associated with loss of CD28 and ?133p53 protein. In poorly proliferative, ?133p53-low CD8+CD28? cells, reconstituted expression of either ?133p53 or CD28 upregulated endogenous expression of each other, which restored cell proliferation, extended replicative lifespan and rescued senescence phenotypes

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    The Journal of Clinical Investigation

  • ISSN

    0021-9738

  • e-ISSN

  • Volume of the periodical

    123

  • Issue of the periodical within the volume

    12

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    11

  • Pages from-to

    5247-5257

  • UT code for WoS article

    000327826100029

  • EID of the result in the Scopus database