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p53-mediated control of gene expression via mRNA translation during Endoplasmic Reticulum stress

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00209805%3A_____%2F15%3A%230000661" target="_blank" >RIV/00209805:_____/15:#0000661 - isvavai.cz</a>

  • Result on the web

    <a href="http://dx.doi.org/10.1080/15384101.2015.1090066" target="_blank" >http://dx.doi.org/10.1080/15384101.2015.1090066</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1080/15384101.2015.1090066" target="_blank" >10.1080/15384101.2015.1090066</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    p53-mediated control of gene expression via mRNA translation during Endoplasmic Reticulum stress

  • Original language description

    p53 is activated by different stress and damage pathways and regulates cell biological responses including cell cycle arrest, repair pathways, apoptosis and senescence. Following DNA damage, the levels of p53 increase and via binding to target gene promoters, p53 induces expression of multiple genes including p21(CDKN1A) and mdm2. The effects of p53 on gene expression during the DNA damage response are well mimicked by overexpressing p53 under normal conditions. However, stress to the Endoplasmic Reticulum (ER) and the consequent Unfolded Protein Response (UPR) leads to the induction of the p53/47 isoform that lacks the first 40 aa of p53 and to an active suppression of p21(CDKN1A) transcription and mRNA translation. We now show that during ER stress p53 also suppresses MDM2 protein levels via a similar mechanism. These observations not only raise questions about the physiological role of MDM2 during ER stress but it also reveals a new facet of p53 as a repressor toward 2 of its major

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/LO1413" target="_blank" >LO1413: RECAMO2020</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2015

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Cell cycle

  • ISSN

    1538-4101

  • e-ISSN

  • Volume of the periodical

    14

  • Issue of the periodical within the volume

    21

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    6

  • Pages from-to

    3373-3378

  • UT code for WoS article

    000364559400011

  • EID of the result in the Scopus database

    2-s2.0-84959491839