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Microbiome and Colorectal Carcinoma Insights From Germ-Free and Conventional Animal Models

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11110%2F14%3A10292189" target="_blank" >RIV/00216208:11110/14:10292189 - isvavai.cz</a>

  • Alternative codes found

    RIV/61388971:_____/14:00435686

  • Result on the web

    <a href="http://dx.doi.org/10.1097/PPO.0000000000000052" target="_blank" >http://dx.doi.org/10.1097/PPO.0000000000000052</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1097/PPO.0000000000000052" target="_blank" >10.1097/PPO.0000000000000052</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Microbiome and Colorectal Carcinoma Insights From Germ-Free and Conventional Animal Models

  • Original language description

    The mammalian microbiota plays a crucial role in the pathogenesis of many diseases. Thanks to recent advances in metagenomics, proteomics, and metabolomics, microbiome composition and metabolic activity can now be studied in detail. Results obtained by such fascinating and provocative studies would be meaningless without considering the perspective of the whole organism. Our work using gnotobiology as the major tool to unravel the mechanisms of host-microbe interaction has demonstrated the crucial roleof microbiota in the initiation and progression of inflammation-associated colorectal neoplasia. Carcinogenesis in the gut is driven by the presence of potentially harmful microbes or by lack of protective ones, by the production of carcinogens generatedby microbes, and by the induction of inflammation and modulation of the immune system. Here, we review these mechanisms with special emphasis on those where gnotobiology has yielded important insights.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EC - Immunology

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    V - Vyzkumna aktivita podporovana z jinych verejnych zdroju

Others

  • Publication year

    2014

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Cancer Journal

  • ISSN

    1528-9117

  • e-ISSN

  • Volume of the periodical

    20

  • Issue of the periodical within the volume

    3

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    8

  • Pages from-to

    217-224

  • UT code for WoS article

    000337708000010

  • EID of the result in the Scopus database