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Substituents at the C3' and C3'N positions are critical for taxanes to overcome acquired resistance of cancer cells to paclitaxel

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11120%2F18%3A43916572" target="_blank" >RIV/00216208:11120/18:43916572 - isvavai.cz</a>

  • Alternative codes found

    RIV/61388963:_____/18:00490321

  • Result on the web

    <a href="https://doi.org/10.1016/j.taap.2018.04.002" target="_blank" >https://doi.org/10.1016/j.taap.2018.04.002</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.taap.2018.04.002" target="_blank" >10.1016/j.taap.2018.04.002</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Substituents at the C3' and C3'N positions are critical for taxanes to overcome acquired resistance of cancer cells to paclitaxel

  • Original language description

    We tested the role of substituents at the C3&apos; and C3&apos;N positions of the taxane molecule to identify taxane derivatives capable of overcoming acquired resistance to paclitaxel. Paclitaxel-resistant sublines SK-BR-3/PacR and MCF-7/PacR as well as the original paclitaxel-sensitive breast cancer cell lines SK-BR-3 and MCF-7 were used for testing. Increased expression of the ABCB1 transporter was found to be involved in the acquired resistance. We tested three groups of taxane derivatives: (1) phenyl group at both C3&apos; and C3&apos;N positions, (2) one phenyl at one of the C3&apos; and C3&apos;N positions and a non-aromatic group at the second position, (3) a non-aromatic group at both C3&apos; and C3&apos;N positions. We found that the presence of phenyl groups at both C3&apos; and C3&apos;N positions is associated with low capability of overcoming acquired paclitaxel resistance compared to taxanes containing at least one non-aromatic substituent at the C3&apos; and C3&apos;N positions. The increase in the ATPase activity of ABCB1 transporter after the application of taxanes from the first group was found to be somewhat higher than after the application of taxanes from the third group. Molecular docking studies demonstrated that the docking score was the lowest, i.e. the highest binding affinity, for taxanes from the first group. It was intermediate for taxanes from the second group, and the highest for taxanes from the third group. We conclude that at least one non-aromatic group at the C3&apos; and C3&apos;N positions of the taxane structure, resulting in reduced affinity to the ABCB1 transporter, brings about high capability of taxane to overcome acquired resistance of breast cancer cells to paclitaxel, due to less efficient transport of the taxane compound out of the cancer cells.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10608 - Biochemistry and molecular biology

Result continuities

  • Project

    <a href="/en/project/LH14096" target="_blank" >LH14096: Structural and functional analysis of taxane derivates capable of overcoming developed resistance to classical taxanes in breast cancer cells</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2018

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Toxicology and Applied Pharmacology

  • ISSN

    0041-008X

  • e-ISSN

  • Volume of the periodical

    347

  • Issue of the periodical within the volume

    May

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    13

  • Pages from-to

    79-91

  • UT code for WoS article

    000431165000009

  • EID of the result in the Scopus database

    2-s2.0-85045191444