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Caffeic acid does not protect human pancreatic β cells against stearic acid-induced ER stress and apoptosis

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11120%2F24%3A43926915" target="_blank" >RIV/00216208:11120/24:43926915 - isvavai.cz</a>

  • Result on the web

    <a href="https://doi.org/10.1002/ejlt.202300206" target="_blank" >https://doi.org/10.1002/ejlt.202300206</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/ejlt.202300206" target="_blank" >10.1002/ejlt.202300206</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Caffeic acid does not protect human pancreatic β cells against stearic acid-induced ER stress and apoptosis

  • Original language description

    Long-term elevation of saturated fatty acids in blood has a deleterious effect on pancreatic beta-cell function and survival, leading to endoplasmic reticulum (ER) stress and apoptosis. This fundamentally contributes to type 2 diabetes development. Caffeic acid (CA) was found to protect various cell types against several proapoptotic stimuli, including fatty acids. However, its potential protective effect against fatty acid-induced apoptosis was not ascertained in pancreatic beta cells yet. Therefore, the objective of this study was to examine this in the human pancreatic beta-cell lines NES2Y and 1.1B4. In both cell lines, CA did not modify the effect of saturated stearic acid (SA) on beta-cell growth and viability. At higher concentrations, CA significantly even intensified the adverse effect of SA. Consistent with this, CA did not exhibit any inhibitory effect on SA-induced markers of ongoing apoptosis as well as ER stress. At higher concentrations, CA again slightly potentiated the effect of SA. CA applied alone was well tolerated up to 1 mM; however, at higher concentrations, it had detrimental effects in both cell lines. To conclude, we have shown that the treatment with caffeic acid has no inhibitory effect on SA-induced ER stress and apoptosis in the human pancreatic beta cells. Moreover, at higher concentrations, CA has proapoptotic potential.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30202 - Endocrinology and metabolism (including diabetes, hormones)

Result continuities

  • Project

    <a href="/en/project/LX22NPO5104" target="_blank" >LX22NPO5104: National Institute for Research of Metabolic and Cardiovascular Diseases</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    European Journal of Lipid Science and Technology

  • ISSN

    1438-7697

  • e-ISSN

    1438-9312

  • Volume of the periodical

    126

  • Issue of the periodical within the volume

    6

  • Country of publishing house

    DE - GERMANY

  • Number of pages

    5

  • Pages from-to

    2300206

  • UT code for WoS article

    001197065700001

  • EID of the result in the Scopus database

    2-s2.0-85190394651