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FOXP1-related intellectual disability syndrome: a recognisable entity

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11130%2F17%3A10373765" target="_blank" >RIV/00216208:11130/17:10373765 - isvavai.cz</a>

  • Alternative codes found

    RIV/00064203:_____/17:10373765

  • Result on the web

    <a href="https://doi.org/10.1136/jmedgenet-2017-104579" target="_blank" >https://doi.org/10.1136/jmedgenet-2017-104579</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1136/jmedgenet-2017-104579" target="_blank" >10.1136/jmedgenet-2017-104579</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    FOXP1-related intellectual disability syndrome: a recognisable entity

  • Original language description

    Background Mutations in forkhead box protein P1 (FOXP1) cause intellectual disability (ID) and specific language impairment (SLI), with or without autistic features (MIM: 613670). Despite multiple case reports no specific phenotype emerged so far. Methods We correlate clinical and molecular data of 25 novel and 23 previously reported patients with FOXP1 defects. We evaluated FOXP1 activity by an in vitro luciferase model and assessed protein stability in vitro by western blotting. Results Patients show ID, SLI, neuromotor delay (NMD) and recurrent facial features including a high broad forehead, bent downslanting palpebral fissures, ptosis and/or blepharophimosis and a bulbous nasal tip. Behavioural problems and autistic features are common. Brain, cardiac and urogenital malformations can be associated. More severe ID and NMD, sensorineural hearing loss and feeding difficulties are more common in patients with interstitial 3p deletions (14 patients) versus patients with monogenic FOXP1 defects (34 patients). Mutations result in impaired transcriptional repression and/or reduced protein stability. Conclusions FOXP1-related ID syndrome is a recognisable entity with a wide clinical spectrum and frequent systemic involvement. Our data will be helpful to evaluate genotype-phenotype correlations when interpreting next-generation sequencing data obtained in patients with ID and/or SLI and will guide clinical management.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10600 - Biological sciences

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2017

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Medical Genetics

  • ISSN

    0022-2593

  • e-ISSN

  • Volume of the periodical

    54

  • Issue of the periodical within the volume

    9

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    11

  • Pages from-to

    613-623

  • UT code for WoS article

    000408150900005

  • EID of the result in the Scopus database

    2-s2.0-85029542030