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Molecular alterations in lesions of anogenital mammary-like glands and their mammary counterparts including hidradenoma papilliferum, intraductal papilloma, fibroadenoma and phyllodes tumor

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11140%2F17%3A10360833" target="_blank" >RIV/00216208:11140/17:10360833 - isvavai.cz</a>

  • Alternative codes found

    RIV/00669806:_____/17:10360833

  • Result on the web

    <a href="http://dx.doi.org/10.1016/j.anndiagpath.2017.02.004" target="_blank" >http://dx.doi.org/10.1016/j.anndiagpath.2017.02.004</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.anndiagpath.2017.02.004" target="_blank" >10.1016/j.anndiagpath.2017.02.004</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Molecular alterations in lesions of anogenital mammary-like glands and their mammary counterparts including hidradenoma papilliferum, intraductal papilloma, fibroadenoma and phyllodes tumor

  • Original language description

    Lesions affecting anogenital mammary-like glands (AGMLG) are histopathologically very similar to those seen in the breast butwhether this morphological similarity is also reflected at the genetic level is unknown. To compare the underlying molecular mechanisms in lesions of AGMLG and their mammary counterparts, we analyzed the mutational profile of 16 anogenital neoplasms including 5 hidradenomas papilliferum (HP), 1 lesion with features of HP and fibroadenoma (FA), 7 FA, 3 phyllodes tumors (PhT)) and 18 analogous breast lesions (6 intraductal papillomas (IDP), 9 FA, and 3 PhT) by high-coverage next generation sequencing (NGS) using a panel comprising 50 cancer-related genes. Additionally, all cases were analyzed for the presence of a mutation in the MED12 gene. All detected mutations with allele frequencies over 20% were independently validated by Sanger sequencing (concordance: 100%). Mutations in PIK3CA, AKT1,MET, ABL1 and TP53 geneswere found in lesions of AGMLG and also their mammary counterparts. The PI3K-AKT cascade plays a role in tumors arising at both sites. It appears that some histopathologically similar anogenital and breast lesions develop along similar molecular pathways.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30216 - Dermatology and venereal diseases

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2017

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Annals of Diagnostic Pathology

  • ISSN

    1092-9134

  • e-ISSN

  • Volume of the periodical

    28

  • Issue of the periodical within the volume

    3

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    7

  • Pages from-to

    12-18

  • UT code for WoS article

    000405760600003

  • EID of the result in the Scopus database

    2-s2.0-85012187393