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The role of p38 in irinotecan-induced DNA damage and apoptosis of colon cancer cells

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11150%2F13%3A10133987" target="_blank" >RIV/00216208:11150/13:10133987 - isvavai.cz</a>

  • Result on the web

    <a href="http://www.sciencedirect.com/science/article/pii/S0027510713000158" target="_blank" >http://www.sciencedirect.com/science/article/pii/S0027510713000158</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.mrfmmm.2013.02.002" target="_blank" >10.1016/j.mrfmmm.2013.02.002</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    The role of p38 in irinotecan-induced DNA damage and apoptosis of colon cancer cells

  • Original language description

    The role of p38 in irinotecan (CPT-11)-induced damage and cell death in colon cancer cell line SW620 was investigated. We demonstrate that CPT-11 treatment activates p38 in exposed cells, however with concentration dependent dynamics and differing consequences. Higher CPT-11 concentrations induce a massive early but relatively short-lasting p38 activity leading to apoptosis mediated by mitochondria and caspases. Pharmacological or siRNA inhibition of p38 then significantly prevents CPT-11-dependent celldeath. Conversely, lower CPT-11 concentrations activate p38 in a delayed, however sustained manner, with apoptosis occurring only in a fraction of cells and in the absence of significant autophagy. Blocking p38 in thus treated cells increases their sensitivity toward CPT-11 and increases cell death. In summary, our results confirm the involvement of p38 in colon cancer cells response to CPT-11 while indicating a varying role of p38 in the final biological response.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Mutation Research

  • ISSN

    0027-5107

  • e-ISSN

  • Volume of the periodical

    741-742

  • Issue of the periodical within the volume

    January - February

  • Country of publishing house

    NL - THE KINGDOM OF THE NETHERLANDS

  • Number of pages

    8

  • Pages from-to

    27-34

  • UT code for WoS article

    000318456400004

  • EID of the result in the Scopus database