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Tirabrutinib-anthracycline interactions in the brain tumor microenvironment: a focus on metabolic inactivation and resistance

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11160%2F25%3A10509886" target="_blank" >RIV/00216208:11160/25:10509886 - isvavai.cz</a>

  • Result on the web

    <a href="https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=_g7jsL2m0r" target="_blank" >https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=_g7jsL2m0r</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1080/17425255.2025.2595661" target="_blank" >10.1080/17425255.2025.2595661</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Tirabrutinib-anthracycline interactions in the brain tumor microenvironment: a focus on metabolic inactivation and resistance

  • Original language description

    Background: Anthracyclines (ANTs) remain essential in many anticancer regimens, yet their clinical utility is often limited by multidrug resistance. Recent studies suggest that Bruton&apos;s tyrosine kinase inhibitors (BTKis) may enhance the efficacy of ANT-based therapies, including those targeting brain tumors. This study investigates how tirabrutinib may contribute to such enhancement via interactions with aldo-keto reductase 1C3 (AKR1C3) and ABC efflux transporters. Research design and methods: The inhibitory potential of tirabrutinib was evaluated using recombinant enzymes, transiently transfected HCT116 cells, and cell lines with differential AKR1C3 expression (T98G, MRC-5). ABC transporter-expressing cells were used to assess daunorubicin accumulation. Western blotting was used to examine whether tirabrutinib alters AKR1C3 protein expression. Results:Tirabrutinib inhibited daunorubicin metabolism in both recombinant AKR1C3 assays and AKR1C3-expressing cells, but had no effect in non-expressing cells. It also enhanced intracellular accumulation of daunorubicin in relevant models. No significant changes in AKR1C3 expression were observed following tirabrutinib treatment. Conclusions: Our in vitro data demonstrate that tirabrutinib influences daunorubicin pharmacodynamics by targeting both metabolic and transport pathways. However, Chou-Talalay analysis highlights the importance of appropriate dosing to achieve therapeutic synergy in combination regimens.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30104 - Pharmacology and pharmacy

Result continuities

  • Project

    <a href="/en/project/EH22_008%2F0004607" target="_blank" >EH22_008/0004607: New Technologies for Translational Research in Pharmaceutical Sciences /NETPHARM</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>S - Specificky vyzkum na vysokych skolach<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Expert Opinion on Drug Metabolism &amp; Toxicology

  • ISSN

    1742-5255

  • e-ISSN

    1744-7607

  • Volume of the periodical

    21

  • Issue of the periodical within the volume

    11-12

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    12

  • Pages from-to

    1315-1326

  • UT code for WoS article

    001629080100001

  • EID of the result in the Scopus database

    2-s2.0-105023706658