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Jagged-mediated development and disease: Mechanistic insights and therapeutic implications for Alagille syndrome

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11310%2F24%3A10487183" target="_blank" >RIV/00216208:11310/24:10487183 - isvavai.cz</a>

  • Result on the web

    <a href="https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=P1bFhwZ0M-" target="_blank" >https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=P1bFhwZ0M-</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.ceb.2023.102302" target="_blank" >10.1016/j.ceb.2023.102302</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Jagged-mediated development and disease: Mechanistic insights and therapeutic implications for Alagille syndrome

  • Original language description

    Notch signaling controls multiple aspects of embryonic development and adult homeostasis. Alagille syndrome is usually caused by a single mutation in the jagged canonical Notch ligand 1 (JAG1), and manifests with liver disease and cardiovascular symptoms that are a direct consequence of JAG1 haploinsufficiency. Recent insights into Jag1/Notch-controlled developmental and homeostatic processes explain how pathology develops in the hepatic and cardiovascular systems and, together with recent elucidation of mechanisms modulating liver regeneration, provide a basis for therapeutic efforts. Importantly, disease presentation can be regulated by genetic modifiers, that may also be therapeutically leverageable. Here, we summarize recent insights into how Jag1 controls processes of relevance to Alagille syndrome, focused on Jag1/ Notch functions in hepatic and cardiovascular development and homeostasis.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10605 - Developmental biology

Result continuities

  • Project

    <a href="/en/project/EH22_010%2F0002902" target="_blank" >EH22_010/0002902: MSCA Fellowships CZ - Charles University</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>S - Specificky vyzkum na vysokych skolach<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Current Opinion in Cell Biology

  • ISSN

    0955-0674

  • e-ISSN

    1879-0410

  • Volume of the periodical

    86

  • Issue of the periodical within the volume

    únor

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    8

  • Pages from-to

    102302

  • UT code for WoS article

    001154778900001

  • EID of the result in the Scopus database

    2-s2.0-85182188559