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Neurodegeneration in Mesial Temporal Lobe Epilepsy (MTLE) Absence of Alpha-Synuclein and TDP-43 Pathology

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216224%3A14110%2F25%3A00142756" target="_blank" >RIV/00216224:14110/25:00142756 - isvavai.cz</a>

  • Result on the web

  • DOI - Digital Object Identifier

Alternative languages

  • Result language

    angličtina

  • Original language name

    Neurodegeneration in Mesial Temporal Lobe Epilepsy (MTLE) Absence of Alpha-Synuclein and TDP-43 Pathology

  • Original language description

    The growing body of evidence concerning the presence of pathological aggregates of hyperphosphorylated tau protein and beta-amyloid in resection specimens from patients with pharmacoresistant mesial temporal lobe epilepsy (MTLE) is well-documented. Recent studies have also suggested alterations in serum levels of alpha-synuclein in this population1,2. Another protein associated with neurodegeneration, TAR DNA-binding protein 43 (TDP-43), has not yet been extensively studied in epilepsy patients. Given this background, the present study aims to investigate the presence of alphasynuclein and TDP-43 pathological aggregates in tissue samples from patients with MTLE sourced from two comprehensive tertiary epilepsy centers

  • Czech name

  • Czech description

Classification

  • Type

    O - Miscellaneous

  • CEP classification

  • OECD FORD branch

    30210 - Clinical neurology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů