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Calcium Signaling in Liver Injury and Regeneration

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216224%3A14310%2F18%3A00113212" target="_blank" >RIV/00216224:14310/18:00113212 - isvavai.cz</a>

  • Result on the web

    <a href="http://dx.doi.org/10.3389/fmed.2018.00192" target="_blank" >http://dx.doi.org/10.3389/fmed.2018.00192</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3389/fmed.2018.00192" target="_blank" >10.3389/fmed.2018.00192</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Calcium Signaling in Liver Injury and Regeneration

  • Original language description

    The liver fulfills central roles in metabolic control and detoxification and, as such, is continuously exposed to a plethora of insults. Importantly, the liver has a unique ability to regenerate and can completely recoup from most acute, non-iterative insults. However, multiple conditions, including viral hepatitis, non-alcoholic fatty liver disease (NAFLD), long-term alcohol abuse and chronic use of certain medications, can cause persistent injury in which the regenerative capacity eventually becomes dysfunctional, resulting in hepatic scaring and cirrhosis. Calcium is a versatile secondary messenger that regulates multiple hepatic functions, including lipid and carbohydrate metabolism, as well as bile secretion and choleresis. Accordingly, dysregulation of calcium signaling is a hallmark of both acute and chronic liver diseases. In addition, recent research implicates calcium transients as essential components of liver regeneration. In this review, we provide a comprehensive overview of the role of calcium signaling in liver health and disease and discuss the importance of calcium in the orchestration of the ensuing regenerative response. Furthermore, we highlight similarities and differences in spatiotemporal calcium regulation between liver insults of different etiologies. Finally, we discuss intracellular calcium control as an emerging therapeutic target for liver injury and summarize recent clinical findings of calcium modulation for the treatment of ischemic-reperfusion injury, cholestasis and NAFLD.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30200 - Clinical medicine

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2018

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    FRONTIERS IN MEDICINE

  • ISSN

    2296-858X

  • e-ISSN

    2296-858X

  • Volume of the periodical

    5

  • Issue of the periodical within the volume

    JUL

  • Country of publishing house

    SE - SWEDEN

  • Number of pages

    17

  • Pages from-to

    1-17

  • UT code for WoS article

    000437327900001

  • EID of the result in the Scopus database

    2-s2.0-85062732746