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Impact of autoimmune gastritis on chronic urticaria in paediatric patients - pathophysiological point of views

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00669806%3A_____%2F24%3A10470922" target="_blank" >RIV/00669806:_____/24:10470922 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11140/24:10470922

  • Result on the web

    <a href="https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=b7kLHdso~K" target="_blank" >https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=b7kLHdso~K</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1007/s00431-023-05324-2" target="_blank" >10.1007/s00431-023-05324-2</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Impact of autoimmune gastritis on chronic urticaria in paediatric patients - pathophysiological point of views

  • Original language description

    We would like to provide an updated comprehensive perspective and identify the components linked to chronic spontaneous urticaria (CSU) without specific triggers in autoimmune atrophic gastritis (AAG). AAG is an organ-specific autoimmune disease that affects the corpus-fundus gastric mucosa. Although we lack a unified explanation of the underlying pathways, when considering all paediatric patients reported in the literature, alterations reslult in gastric neuroendocrine enterochrmaffin-like (ECL) cell proliferation and paracrine release of histamine. Several mechanism have been proposed for the pathogenesis of CSU, with much evidence pointing towards AAG and ECL cell responses, which may be implicated as potential factors contributing to CSU. The excessive production/release of histamine into the bloodstream could cause or trigger exacerbations of CSU in AAG, independent of Helicobacter pylori; thus, the release of histamine from ECL cells may be the primary modulator. Conclusion: Considering the understanding of these interactions, recognising the respective roles of AAG in the pathogenesis of CSU may strongly impactt the diagnostic workup and management of unexplained/refractory CSU and may inform future research and interventions in the paediatric population.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30209 - Paediatrics

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    European Journal of Pediatrics

  • ISSN

    0340-6199

  • e-ISSN

    1432-1076

  • Volume of the periodical

    183

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    DE - GERMANY

  • Number of pages

    8

  • Pages from-to

    515-522

  • UT code for WoS article

    001100533400001

  • EID of the result in the Scopus database

    2-s2.0-85176545925