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SAL1-PAP retrograde signaling orchestrates photosynthetic and extracellular reactive oxygen species for stress responses

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F60076658%3A12310%2F25%3A43910098" target="_blank" >RIV/60076658:12310/25:43910098 - isvavai.cz</a>

  • Result on the web

    <a href="https://onlinelibrary.wiley.com/doi/epdf/10.1111/tpj.70271?getft_integrator=clarivate&src=getftr&utm_source=clarivate" target="_blank" >https://onlinelibrary.wiley.com/doi/epdf/10.1111/tpj.70271?getft_integrator=clarivate&src=getftr&utm_source=clarivate</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1111/tpj.70271" target="_blank" >10.1111/tpj.70271</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    SAL1-PAP retrograde signaling orchestrates photosynthetic and extracellular reactive oxygen species for stress responses

  • Original language description

    Cellular responses to abiotic stress involve multiple signals such as reactive oxygen species (ROS), Ca2+, abscisic acid (ABA), and chloroplast-to-nucleus retrograde signals such as 3 &apos;-phosphoadenosine 5 &apos;-phosphate (PAP). The mechanism(s) by which these messengers intersect for cell regulation remain enigmatic, as do the roles of retrograde signals in specialized cells. Here we demonstrate a mechanistic link enabling ABA and PAP to coordinate chloroplast and plasma membrane ROS production. Contrary to its role in upregulating processes leading to quenching of ROS in foliar tissue, we show that in guard cells, PAP induces chloroplast ROS accumulation via photosynthetic electron transport and apoplast ROS via the RESPIRATORY BURST OXIDASE HOMOLOG (RBOH) proteins. Both subcellular ROS sources are necessary for stress hormone ABA-mediated stomatal closure, as well as PAP-mediated stomatal closure. However, PAP signaling diverges from ABA by activating RBOHD instead of RBOHF. Three calcium-dependent protein kinases (CPKs) transcriptionally induced by PAP, namely CPK13, CPK32, and CPK34, concurrently activate RBOHD and the slow anion channel SLAC1 by phosphorylating two SLAC1 serine (S) residues, including S120, which is also targeted by the ABA signaling kinase OPEN STOMATA 1 (OST1). Consequently, overexpression of the PAP-induced CPKs rescues stomatal closure in ost1. Our data identify chloroplast retrograde signals as critical nodes in cellular stress response networks of guard cells.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10611 - Plant sciences, botany

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Plant Journal

  • ISSN

    0960-7412

  • e-ISSN

    1365-313X

  • Volume of the periodical

    122

  • Issue of the periodical within the volume

    5

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    16

  • Pages from-to

    nestránkováno

  • UT code for WoS article

    001508964000004

  • EID of the result in the Scopus database

    2-s2.0-105008277891