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Chelation of Mitochondrial Iron as an Antiparasitic Strategy

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F60077344%3A_____%2F24%3A00602376" target="_blank" >RIV/60077344:_____/24:00602376 - isvavai.cz</a>

  • Alternative codes found

    RIV/86652036:_____/24:00602376 RIV/00023001:_____/24:00084584 RIV/00216208:11310/24:10479589

  • Result on the web

    <a href="https://pubs.acs.org/doi/epdf/10.1021/acsinfecdis.3c00529?ref=article_openPDF" target="_blank" >https://pubs.acs.org/doi/epdf/10.1021/acsinfecdis.3c00529?ref=article_openPDF</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1021/acsinfecdis.3c00529" target="_blank" >10.1021/acsinfecdis.3c00529</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Chelation of Mitochondrial Iron as an Antiparasitic Strategy

  • Original language description

    Iron, as an essential micronutrient, plays a crucial role in host-pathogen interactions. In order to limit the growth of the pathogen, a common strategy of innate immunity includes withdrawing available iron to interfere with the cellular processes of the microorganism. Against that, unicellular parasites have developed powerful strategies to scavenge iron, despite the effort of the host. Iron-sequestering compounds, such as the approved and potent chelator deferoxamine (DFO), are considered a viable option for therapeutic intervention. Since iron is heavily utilized in the mitochondrion, targeting iron chelators in this organelle could constitute an effective therapeutic strategy. This work presents mitochondrially targeted DFO, mitoDFO, as a candidate against a range of unicellular parasites with promising in vitro efficiency. Intracellular Leishmania infection can be cleared by this compound, and experimentation with Trypanosoma brucei 427 elucidates its possible mode of action. The compound not only affects iron homeostasis but also alters the physiochemical properties of the inner mitochondrial membrane, resulting in a loss of function. Furthermore, investigating the virulence factors of pathogenic yeasts confirms that mitoDFO is a viable candidate for therapeutic intervention against a wide spectrum of microbe-associated diseases.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10606 - Microbiology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    ACS Infectious Diseases

  • ISSN

    2373-8227

  • e-ISSN

  • Volume of the periodical

    10

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    12

  • Pages from-to

    676-687

  • UT code for WoS article

    001161279500001

  • EID of the result in the Scopus database

    2-s2.0-85184834212