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A genetically encoded nanobody sensor reveals conformational diversity in β-arrestins orchestrated by distinct seven transmembrane receptors

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61388963%3A_____%2F25%3A00639402" target="_blank" >RIV/61388963:_____/25:00639402 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11110/25:10503588

  • Result on the web

    <a href="https://doi.org/10.1073/pnas.2507384122" target="_blank" >https://doi.org/10.1073/pnas.2507384122</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1073/pnas.2507384122" target="_blank" >10.1073/pnas.2507384122</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    A genetically encoded nanobody sensor reveals conformational diversity in β-arrestins orchestrated by distinct seven transmembrane receptors

  • Original language description

    Agonist-induced interaction of G protein-coupled receptors (GPCRs) with beta- arrestins (beta arrs) is a critical mechanism that regulates the spatiotemporal pattern of receptor localization and signaling. While the underlying mechanism governing GPCR-beta arr interaction is primarily conserved and involves receptor activation and phosphorylation, there are several examples of receptor-specific fine-tuning of beta arr-mediated functional outcomes. Considering the key contribution of conformational plasticity of beta arrs in driving receptor-specific functional responses, it is important to develop novel sensors capable of reporting distinct beta arr conformations in cellular context. Here, we design an intrabody version of a beta arr-recognizing nanobody (nanobody32), referred to as intrabody32 (Ib32), in NanoLuc enzyme complementation assay format and measure its ability to recognize beta arr1 and 2 in live cells upon activation of a broad set of GPCRs. Ib32 robustly recognizes activated beta arr1 and 2 in the plasma membrane and endosomes, and effectively mirrors beta arr recruitment profile upon stimulation of selected GPCRs. We also design an Ib32 sensor for polarization microscopy with a change in linear dichroism as readout and demonstrate its utility for monitoring beta arr activation upon stimulation of selected GPCRs by natural and biased agonists. Taken together with a previously described sensor of beta arr1 activation, Ib32 underscores the inherent flexibility encoded in beta arrs and conformational diversity imparted by different GPCRs, which is further corroborated using an orthogonal limited proteolysis assay. Our study presents Ib32 as a sensor of beta arr activation and highlights the structural diversity of beta arrs, which likely allows their ability to interact with, and regulate, a large repertoire of GPCRs.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10610 - Biophysics

Result continuities

  • Project

    <a href="/en/project/LTAIN19167" target="_blank" >LTAIN19167: Visualizing GPCR-G-protein-beta-arrestin supercomplexes in living cells using two-photon polarization microscopy</a><br>

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Proceedings of the National Academy of Sciences of the United States of America

  • ISSN

    0027-8424

  • e-ISSN

    1091-6490

  • Volume of the periodical

    122

  • Issue of the periodical within the volume

    37

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    12

  • Pages from-to

    e2507384122

  • UT code for WoS article

    001576021100001

  • EID of the result in the Scopus database

    2-s2.0-105015671723