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Epigenetic targeting of transposon relics: beating the dead horses of the genome?

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61389030%3A_____%2F22%3A00561925" target="_blank" >RIV/61389030:_____/22:00561925 - isvavai.cz</a>

  • Alternative codes found

    RIV/67985939:_____/22:00561925 RIV/00216208:11310/22:10445403

  • Result on the web

    <a href="https://doi.org/10.1080/15592294.2021.2022066" target="_blank" >https://doi.org/10.1080/15592294.2021.2022066</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1080/15592294.2021.2022066" target="_blank" >10.1080/15592294.2021.2022066</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Epigenetic targeting of transposon relics: beating the dead horses of the genome?

  • Original language description

    Transposable elements (TEs) have been seen as selfish genetic elements that can propagate in a host genome. Their propagation success is however hindered by a combination of mechanisms such as mutations, selection, and their epigenetic silencing by the host genome. As a result, most copies of TEs in a given genome are dead relics: their sequence is too degenerated to allow any transposition. Nevertheless, these TE relics often, but not always, remain epigenetically silenced, and if not to prevent transposition anymore, one can wonder the reason for this phenomenon. The mere self-perpetuating loop inherent to epigenetic silencing could alone explain that even when inactive, TE copies remain silenced. Beyond this process, nevertheless, antagonistic selective forces are likely to act on TE relic silencing. Especially, without the benefit of preventing transposition, TE relic silencing may prove deleterious to the host fitness, suggesting that the maintenance of TE relic silencing is the result of a fine, and perhaps case-by-case, evolutionary trade-off between beneficial and deleterious effects. Ultimately, the release of TE relics silencing may provide a ‘safe’ ground for adaptive epimutations to arise. In this review, we provide an overview of these questions in both plants and animals.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10603 - Genetics and heredity (medical genetics to be 3)

Result continuities

  • Project

    <a href="/en/project/GA22-20240S" target="_blank" >GA22-20240S: Deciphering the biological significance of genomic imprinting</a><br>

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2022

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Epigenetics

  • ISSN

    1559-2294

  • e-ISSN

    1559-2308

  • Volume of the periodical

    17

  • Issue of the periodical within the volume

    11

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    14

  • Pages from-to

    1331-1344

  • UT code for WoS article

    000738476000001

  • EID of the result in the Scopus database

    2-s2.0-85122318163