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Crohn's disease - genetic factors and progress of the disease.

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61988987%3A17110%2F18%3AA1901SMQ" target="_blank" >RIV/61988987:17110/18:A1901SMQ - isvavai.cz</a>

  • Alternative codes found

    RIV/61989100:27640/18:10237217 RIV/00843989:_____/18:E0107133

  • Result on the web

    <a href="http://dx.doi.org/10.5507/bp.2017.058" target="_blank" >http://dx.doi.org/10.5507/bp.2017.058</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.5507/bp.2017.058" target="_blank" >10.5507/bp.2017.058</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Crohn's disease - genetic factors and progress of the disease.

  • Original language description

    Background and Objectives. Crohn?s disease is a multifactorial inflammatory disease affecting mainly the gastrointestinaltract. The genetic factors that are involved in the disease include mainly three mutations of the gene NOD2/CARD15 (R702W, G908R, 3020insC). The aim of this study was to determine the relationship between the presence ofthese variants and disease phenotype.Material and Methods. 70 patients with Crohn?s disease were examined for the presence of the above-mentionedmutations. The researchers used the medical records to retrospectively obtain clinical data and together with the informationobtained prospectively according to the protocol they analysed the connection between gene mutationsand disease phenotype.Results. At least one mutation was found in 22 patients with Crohn?s disease (32%), four patients were found to havetwo different mutations (composed heterozygotes - 6%) and six patients (9%) were homozygotes for the 3020insCgene. No significant differences were found between the groups with wild-type form and the mutated form of theNOD2 / CARD15 gene with respect to age at the time of diagnosis, form of the disease or localization according to theMontreal classification.Conclusion. Mutations of the NOD2 / CARD15 gene did not significantly affect the frequency of reoperations, homozygoteswith 3020insC gene mutations, however, represented a high risk group. The phenotype was not relatedsignificantly to the presence of the examined mutations.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30219 - Gastroenterology and hepatology

Result continuities

  • Project

  • Continuities

    V - Vyzkumna aktivita podporovana z jinych verejnych zdroju

Others

  • Publication year

    2018

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Biomedical Papers

  • ISSN

    1213-8118

  • e-ISSN

    1804-7521

  • Volume of the periodical

    2

  • Issue of the periodical within the volume

    162

  • Country of publishing house

    CZ - CZECH REPUBLIC

  • Number of pages

    5

  • Pages from-to

    139-143

  • UT code for WoS article

    000436347500011

  • EID of the result in the Scopus database

    2-s2.0-85049067609