All

What are you looking for?

All
Projects
Results
Organizations

Quick search

  • Projects supported by TA ČR
  • Excellent projects
  • Projects with the highest public support
  • Current projects

Smart search

  • That is how I find a specific +word
  • That is how I leave the -word out of the results
  • “That is how I can find the whole phrase”

Nucleoporin NUP153 guards genome integrity by promoting nuclear import of 53BP1

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61989592%3A15110%2F12%3A33140851" target="_blank" >RIV/61989592:15110/12:33140851 - isvavai.cz</a>

  • Alternative codes found

    RIV/68378050:_____/12:00387882

  • Result on the web

    <a href="http://dx.doi.org/10.1038/cdd.2011.150" target="_blank" >http://dx.doi.org/10.1038/cdd.2011.150</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/cdd.2011.150" target="_blank" >10.1038/cdd.2011.150</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Nucleoporin NUP153 guards genome integrity by promoting nuclear import of 53BP1

  • Original language description

    53BP1 is a mediator of DNA damage response (DDR) and a tumor suppressor whose accumulation on damaged chromatin promotes DNA repair and enhances DDR signaling. Using foci formation of 53BP1 as a readout in two human cell lines, we performed an siRNA-based functional high-content microscopy screen for modulators of cellular response to ionizing radiation (IR). Here, we provide the complete results of this screen as an information resource, and validate and functionally characterize one of the identified'hits': a nuclear pore component NUP153 as a novel factor specifically required for 53BP1 nuclear import. Using a range of cell and molecular biology approaches including live-cell imaging, we show that knockdown of NUP153 prevents 53BP1, but not severalother DDR factors, from entering the nuclei in the newly forming daughter cells. This translates into decreased IR-induced 53BP1 focus formation, delayed DNA repair and impaired cell survival after IR. In addition, NUP153 depletion exace

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    FD - Oncology and haematology

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2012

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    CELL DEATH AND DIFFERENTIATION

  • ISSN

    1350-9047

  • e-ISSN

  • Volume of the periodical

    19

  • Issue of the periodical within the volume

    5

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    10

  • Pages from-to

    798-807

  • UT code for WoS article

    000302784400008

  • EID of the result in the Scopus database