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STAT6 - polymorphisms, haplotypes and epistasis in relation to atopy and asthma

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61989592%3A15110%2F13%3A33145486" target="_blank" >RIV/61989592:15110/13:33145486 - isvavai.cz</a>

  • Alternative codes found

    RIV/00098892:_____/13:#0000530

  • Result on the web

    <a href="http://dx.doi.org/10.5507/bp.2013.043" target="_blank" >http://dx.doi.org/10.5507/bp.2013.043</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.5507/bp.2013.043" target="_blank" >10.5507/bp.2013.043</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    STAT6 - polymorphisms, haplotypes and epistasis in relation to atopy and asthma

  • Original language description

    Background. STAT6 has an important role in the IL-4/IL-13 signalling pathway. Genome - wide association studies have shown that particular polymorphism (SNP) or haplorype variants of STAT6 as well as epigenetic gene modifications are associated with IgElevel and astma in childhood. Methods. A review of the available literature was performed to map out the function and signalling patway of STAT6, studies of STAT6 SNP s association with susceptibility to asthma and atopy, covering the years 1997 - 2012 were summarized, and the value of epigenetic and epistatic influences on STAT6 and their relevance to the development of the studiend phenotype (atopy or asthma) were determined. Results. There are 2 SNPs (rs71802646 and rs320411) with clinical association and proven functional effect on STAT6 expression. The effect of STAT6 SNPs cumulates in haplotypes and more potently during interaction with SNPs in the genes from the signallling pathway (IL4, IL4Ra, and IL13). Expression of STAT6 is o

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Biomedical Papers-Olomouc

  • ISSN

    1213-8118

  • e-ISSN

  • Volume of the periodical

    157

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    CZ - CZECH REPUBLIC

  • Number of pages

    9

  • Pages from-to

    172-180

  • UT code for WoS article

  • EID of the result in the Scopus database