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Cortical somatosensory processing after botulinum toxin therapy in post-stroke spasticity

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F61989592%3A15110%2F21%3A73607908" target="_blank" >RIV/61989592:15110/21:73607908 - isvavai.cz</a>

  • Alternative codes found

    RIV/00098892:_____/21:N0000054

  • Result on the web

    <a href="https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8238289/pdf/medi-100-e26356.pdf" target="_blank" >https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8238289/pdf/medi-100-e26356.pdf</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1097/MD.0000000000026356" target="_blank" >10.1097/MD.0000000000026356</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Cortical somatosensory processing after botulinum toxin therapy in post-stroke spasticity

  • Original language description

    Abstract In dystonic and spastic movement disorders, abnormalities of motor control and somatosensory processing as well as cortical modulations associated with clinical improvement after botulinum toxin A (BoNT-A) treatment have been reported, but electrophysiological evidence remains controversial. In the present observational study, we aimed to uncover central correlates of post-stroke spasticity (PSS) and BoNT-A-related changes in the sensorimotor cortex by investigating the cortical components of somatosensory evoked potentials (SEPs). Thirty-one chronic stroke patients with PSS of the upper limb were treated with BoNT-A application into the affected muscles and physiotherapy. Clinical and electrophysiological evaluations were performed just before BoNT-A application (W0), then 4 weeks (W4) and 11weeks (W11) later. PSS was evaluated with the modified Ashworth scale (MAS). Median nerve SEPs were examined in both upper limbs with subsequent statistical analysis of the peak-to-peak amplitudes of precentral P22/N30 and postcentral N20/P23 components. At baseline (W0), postcentral SEPs were significantly lower over the affected cortex. At follow up, cortical SEPs did not show any significant changes attributable to BoNT-A and/or physiotherapy, despite clear clinical improvement. Our results imply that conventional SEPs are of limited value in evaluating cortical changes after BoNT-A treatment and further studies are needed to elucidate its central actions. Abbreviations: ADL = activities of daily living, BoNT-A = botulinum toxin A, CNS = central nervous system, fMRI = functional magnetic resonance imaging, IQR = interquartile range, MAS = modified Ashworth scale, PPC = posterior parietal cortex, PSS = post-stroke spasticity, SD = standard deviation, SEPs = somatosensory evoked potentials.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30210 - Clinical neurology

Result continuities

  • Project

    <a href="/en/project/NV17-29452A" target="_blank" >NV17-29452A: Sensorimotor network modulation by targeted therapy of post-stroke spasticity</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    MEDICINE

  • ISSN

    0025-7974

  • e-ISSN

  • Volume of the periodical

    25

  • Issue of the periodical within the volume

    100

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    7

  • Pages from-to

    "nestránkováno"

  • UT code for WoS article

    000664672200028

  • EID of the result in the Scopus database

    2-s2.0-85109017978