Redox Homeostasis in Pancreatic beta Cells

Result code in IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F67985823%3A_____%2F12%3A00389998" target="_blank" >RIV/67985823:_____/12:00389998 - isvavai.cz</a>
Result on the web
<a href="http://dx.doi.org/10.1155/2012/932838" target="_blank" >http://dx.doi.org/10.1155/2012/932838</a>
DOI - Digital Object Identifier
<a href="http://dx.doi.org/10.1155/2012/932838" target="_blank" >10.1155/2012/932838</a>

Result language
angličtina
Original language name
Redox Homeostasis in Pancreatic beta Cells
Original language description
In this article authors reviewed mechanism that determine reactive oxygen species (redox) homeostasis, redox information signaling and metabolic/regulatory function of autocrine insulin signaling in pancreatic beta cells, and consequences of oxidative stress and dysregulation of redox/information signaling for their dysfunction. They emphasized the role of mitochondrion in beta cell molecular physiology and pathology, including the antioxidant role of mitochondrial uncoupling protein 2
Czech name
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Czech description
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Type
J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)
CEP classification
FB - Endocrinology, diabetology, metabolism, nutrition
OECD FORD branch
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Project
Result was created during the realization of more than one project. More information in the Projects tab.
Continuities
I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Publication year
2012
Confidentiality
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

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