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Solving the MCM paradox by visualizing the scaffold of CMG helicase at active replisomes

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68081707%3A_____%2F22%3A00564037" target="_blank" >RIV/68081707:_____/22:00564037 - isvavai.cz</a>

  • Result on the web

    <a href="https://www.nature.com/articles/s41467-022-33887-5" target="_blank" >https://www.nature.com/articles/s41467-022-33887-5</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/s41467-022-33887-5" target="_blank" >10.1038/s41467-022-33887-5</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Solving the MCM paradox by visualizing the scaffold of CMG helicase at active replisomes

  • Original language description

    Genome duplication is safeguarded by constantly adjusting the activity of the replicative CMG (CDC45-MCM2-7-GINS) helicase. However, minichromosome maintenance proteins (MCMs)-the structural core of the CMG helicase-have never been visualized at sites of DNA synthesis inside a cell (the so-called MCM paradox). Here, we solve this conundrum by showing that anti-MCM antibodies primarily detect inactive MCMs. Upon conversion of inactive MCMs to CMGs, factors that are required for replisome activity bind to the MCM scaffold and block MCM antibody binding sites. Tagging of endogenous MCMs by CRISPR-Cas9 bypasses this steric hindrance and enables MCM visualization at active replisomes. Thus, by defining conditions for detecting the structural core of the replicative CMG helicase, our results explain the MCM paradox, provide visual proof that MCMs are an integral part of active replisomes in vivo, and enable the investigation of replication dynamics in living cells exposed to a constantly changing environment.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10601 - Cell biology

Result continuities

  • Project

    <a href="/en/project/GM22-20303M" target="_blank" >GM22-20303M: Deciphering Origins of DNA replication in genome integrity</a><br>

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2022

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Nature Communications

  • ISSN

    2041-1723

  • e-ISSN

    2041-1723

  • Volume of the periodical

    13

  • Issue of the periodical within the volume

    1

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    11

  • Pages from-to

    6090

  • UT code for WoS article

    000874078500024

  • EID of the result in the Scopus database

    2-s2.0-85139886579