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Associations between pancreatic expression quantitative traits and risk of pancreatic ductal adenocarcinoma

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378041%3A_____%2F21%3A00552790" target="_blank" >RIV/68378041:_____/21:00552790 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11120/21:43921764 RIV/00216208:11140/21:10429094 RIV/00216208:11110/21:10429094 RIV/00064173:_____/21:N0000128 RIV/61989592:15110/21:73608705

  • Result on the web

    <a href="https://academic.oup.com/carcin/article-abstract/42/8/1037/6314283?redirectedFrom=fulltext" target="_blank" >https://academic.oup.com/carcin/article-abstract/42/8/1037/6314283?redirectedFrom=fulltext</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1093/carcin/bgab057" target="_blank" >10.1093/carcin/bgab057</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Associations between pancreatic expression quantitative traits and risk of pancreatic ductal adenocarcinoma

  • Original language description

    Pancreatic ductal adenocarcinoma (PDAC) is among the most lethal cancers. Its poor prognosis is predominantly due to the fact that most patients remain asymptomatic until the disease reaches an advanced stage, alongside the lack of early markers and screening strategies. A better understanding of PDAC risk factors is essential for the identification of groups at high risk in the population. Genome-wide association studies (GWAS) have been a powerful tool for detecting genetic variants associated with complex traits, including pancreatic cancer. By exploiting functional and GWAS data, we investigated the associations between polymorphisms affecting gene function in the pancreas (expression quantitative trait loci, eQTLs) and PDAC risk. In a two-phase approach, we analysed 13 713 PDAC cases and 43 784 controls and identified a genome-wide significant association between the A allele of the rs2035875 polymorphism and increased PDAC risk (P = 7.14 x 10(-10)). This allele is known to be associated with increased expression in the pancreas of the keratin genes KRT8 and KRT18, whose increased levels have been reported to correlate with various tumour cell characteristics. Additionally, the A allele of the rs789744 variant was associated with decreased risk of developing PDAC (P = 3.56 x 10(-6)). This single nucleotide polymorphism is situated in the SRGAP1 gene and the A allele is associated with higher expression of the gene, which in turn inactivates the cyclin-dependent protein 42 (CDC42) gene expression, thus decreasing the risk of PDAC. In conclusion, we present here a functional-based novel PDAC risk locus and an additional strong candidate supported by significant associations and plausible biological mechanisms.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10603 - Genetics and heredity (medical genetics to be 3)

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2021

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Carcinogenesis

  • ISSN

    0143-3334

  • e-ISSN

    1460-2180

  • Volume of the periodical

    42

  • Issue of the periodical within the volume

    8

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    9

  • Pages from-to

    1037-1045

  • UT code for WoS article

    000692322000004

  • EID of the result in the Scopus database

    2-s2.0-85114637768