The deletion of AQP4 and TRPV4 affects astrocyte swelling/volume recovery in response to ischemia-mimicking pathologies

Result code in IS VaVaI
<a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378041%3A_____%2F24%3A00585938" target="_blank" >RIV/68378041:_____/24:00585938 - isvavai.cz</a>
Alternative codes found
RIV/86652036:_____/24:00617007 RIV/00216208:11130/24:10480899
Result on the web
<a href="https://www.frontiersin.org/articles/10.3389/fncel.2024.1393751/full" target="_blank" >https://www.frontiersin.org/articles/10.3389/fncel.2024.1393751/full</a>
DOI - Digital Object Identifier
<a href="http://dx.doi.org/10.3389/fncel.2024.1393751" target="_blank" >10.3389/fncel.2024.1393751</a>

Result language
angličtina
Original language name
The deletion of AQP4 and TRPV4 affects astrocyte swelling/volume recovery in response to ischemia-mimicking pathologies
Original language description
Astrocytic Transient receptor potential vanilloid 4 (TRPV4) channels, together with Aquaporin 4 (AQP4), are suspected to be the key players in cellular volume regulation, and therefore may affect the development and severity of cerebral edema during ischemia. In this study, we examined astrocytic swelling/volume recovery in mice with TRPV4 and/or AQP4 deletion in response to in vitro ischemic conditions, to determine how the deletion of these channels can affect the development of cerebral edema.
Czech name
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Czech description
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Type
J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database
CEP classification
—
OECD FORD branch
30103 - Neurosciences (including psychophysiology)

Project
Result was created during the realization of more than one project. More information in the Projects tab.
Continuities
I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Publication year
2024
Confidentiality
S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

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