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A review of standardized high-throughput cardiovascular phenotyping with a link to metabolism in mice

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F68378050%3A_____%2F23%3A00573452" target="_blank" >RIV/68378050:_____/23:00573452 - isvavai.cz</a>

  • Result on the web

    <a href="https://link.springer.com/article/10.1007/s00335-023-09997-w" target="_blank" >https://link.springer.com/article/10.1007/s00335-023-09997-w</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1007/s00335-023-09997-w" target="_blank" >10.1007/s00335-023-09997-w</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    A review of standardized high-throughput cardiovascular phenotyping with a link to metabolism in mice

  • Original language description

    Cardiovascular diseases cause a high mortality rate worldwide and represent a major burden for health care systems. Experimental rodent models play a central role in cardiovascular disease research by effectively simulating human cardiovascular diseases. Using mice, the International Mouse Phenotyping Consortium (IMPC) aims to target each protein-coding gene and phenotype multiple organ systems in single-gene knockout models by a global network of mouse clinics. In this review, we summarize the current advances of the IMPC in cardiac research and describe in detail the diagnostic requirements of high-throughput electrocardiography and transthoracic echocardiography capable of detecting cardiac arrhythmias and cardiomyopathies in mice. Beyond that, we are linking metabolism to the heart and describing phenotypes that emerge in a set of known genes, when knocked out in mice, such as the leptin receptor (Lepr), leptin (Lep), and Bardet–Biedl syndrome 5 (Bbs5). Furthermore, we are presenting not yet associated loss-of-function genes affecting both, metabolism and the cardiovascular system, such as the RING finger protein 10 (Rfn10), F-box protein 38 (Fbxo38), and Dipeptidyl peptidase 8 (Dpp8). These extensive high-throughput data from IMPC mice provide a promising opportunity to explore genetics causing metabolic heart disease with an important translational approach.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    10608 - Biochemistry and molecular biology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2023

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Mammalian Genome

  • ISSN

    0938-8990

  • e-ISSN

    1432-1777

  • Volume of the periodical

    34

  • Issue of the periodical within the volume

    2

  • Country of publishing house

    DE - GERMANY

  • Number of pages

    16

  • Pages from-to

    107-122

  • UT code for WoS article

    001012256500002

  • EID of the result in the Scopus database

    2-s2.0-85162027607