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The mobility of mitochondria: Intercellular trafficking in health and disease

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F86652036%3A_____%2F17%3A00507981" target="_blank" >RIV/86652036:_____/17:00507981 - isvavai.cz</a>

  • Result on the web

    <a href="https://onlinelibrary.wiley.com/doi/full/10.1111/1440-1681.12764" target="_blank" >https://onlinelibrary.wiley.com/doi/full/10.1111/1440-1681.12764</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1111/1440-1681.12764" target="_blank" >10.1111/1440-1681.12764</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    The mobility of mitochondria: Intercellular trafficking in health and disease

  • Original language description

    The view that genes are constrained within somatic cells is challenged by in vitro evidence, and more recently by in vivo studies which demonstrate that mitochondria with their mitochondrial DNA (mtDNA) payload not only can, but do move between cells in tumour models and in mouse models of tissue damage. Using mouse tumour cell models without mtDNA to reflect mtDNA damage, we have shown that these cells grow tumours only after acquiring mtDNA from cells in the local microenvironment resulting in respiration recovery, tumorigenesis and metastasis. Mitochondrial transfer between cells has also been demonstrated following ischaemia-induced injury in the heart and brain and in lung epithelium, and following lung inflammation. In vitro investigations suggest that stem cells may be mitochondrial donors. The ability of mitochondria to move between cells appears to be an evolutionarily-conserved phenomenon, relevant to diseases with compromised mitochondrial function including neurodegenerative, neuromuscular and cardiovascular diseases as well as cancer and ageing.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30104 - Pharmacology and pharmacy

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2017

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Clinical and Experimental Pharmacology and Physiology

  • ISSN

    1440-1681

  • e-ISSN

  • Volume of the periodical

    44

  • Issue of the periodical within the volume

    DEC 2017

  • Country of publishing house

    AU - AUSTRALIA

  • Number of pages

    6

  • Pages from-to

    15-20

  • UT code for WoS article

    000418663800003

  • EID of the result in the Scopus database

    2-s2.0-85030033108