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Atypical hemolytic uremic syndrome triggered by mRNA vaccination against SARS-CoV-2 : case report

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023001%3A_____%2F22%3A00083477" target="_blank" >RIV/00023001:_____/22:00083477 - isvavai.cz</a>

  • Alternative codes found

    RIV/00064165:_____/22:10449969 RIV/00216208:11110/22:10449969 RIV/00216208:11150/22:10449969

  • Result on the web

    <a href="https://www.frontiersin.org/articles/10.3389/fimmu.2022.1001366/full" target="_blank" >https://www.frontiersin.org/articles/10.3389/fimmu.2022.1001366/full</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.3389/fimmu.2022.1001366" target="_blank" >10.3389/fimmu.2022.1001366</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Atypical hemolytic uremic syndrome triggered by mRNA vaccination against SARS-CoV-2 : case report

  • Original language description

    Atypical hemolytic uremic syndrome (aHUS), also called complement-mediated hemolytic uremic syndrome (CM-HUS), is a rare disease caused by dysregulation in the alternative complement activation pathway. It is a life-threatening condition causing ischemia of a number of organs, and it typically causes acute kidney injury. This disorder may be triggered by various factors including viral or bacterial infections, pregnancy, surgery, and injuries. In about 60% of cases, the genetic origin of the disease can be identified-commonly mutations affecting complementary factor H and MCP protein. Eculizumab, a monoclonal antibody to the C5 component of the complement, represents the current effective treatment.We describe a case of a young woman with a previous history of polyvalent allergies, who developed atypical hemolytic uremic syndrome after vaccination with mRNA vaccine against SARS-CoV-2. The disease manifested by scleral bleeding, acute renal insufficiency, anemia, and thrombocytopenia. The patient was treated with plasma exchanges without sufficient effect; remission occurred only after starting treatment with eculizumab. Genetic examination showed that the patient is a carrier of multiple inherited risk factors (a rare pathogenic variant in CFH, MCPggaac haplotype of the CD46 gene, and the risk haplotype CFH H3). The patient is currently in hematological remission with persistent mild renal insufficiency, continuing treatment with eculizumab/ravulizumab. By this case report, we meant to point out the need for careful monitoring of people after vaccination, as it may trigger immune-mediated diseases, especially in those with predisposing factors.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30217 - Urology and nephrology

Result continuities

  • Project

  • Continuities

    V - Vyzkumna aktivita podporovana z jinych verejnych zdroju

Others

  • Publication year

    2022

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Frontiers in immunology [online]

  • ISSN

    1664-3224

  • e-ISSN

    1664-3224

  • Volume of the periodical

    13

  • Issue of the periodical within the volume

    September 28

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    9

  • Pages from-to

    "art. no. 1001366"

  • UT code for WoS article

    000872783800001

  • EID of the result in the Scopus database

    2-s2.0-85140125547