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Exploring the potential of soluble guanylyl cyclase stimulators and activators in heart failure

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023001%3A_____%2F25%3A00085885" target="_blank" >RIV/00023001:_____/25:00085885 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11130/25:10503231 RIV/00064203:_____/25:10503231

  • Result on the web

    <a href="https://www.sciencedirect.com/science/article/pii/S0006295225006288?via%3Dihub" target="_blank" >https://www.sciencedirect.com/science/article/pii/S0006295225006288?via%3Dihub</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.bcp.2025.117363" target="_blank" >10.1016/j.bcp.2025.117363</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Exploring the potential of soluble guanylyl cyclase stimulators and activators in heart failure

  • Original language description

    Heart failure (HF) is a life-threatening disease characterized by substantial morbidity and mortality. Yet despite recent advances, prognosis remains poor. Cyclic guanosine 3 &apos;,5 &apos;-monophosphate (cGMP) mediates a wide range of physiological processes in various cell types. Its deficiency has been implicated in numerous pathological cardiovascular diseases, including HF, pulmonary hypertension (PH), and kidney disease. Therefore, restoring and enhancing the nitric oxide (NO)-soluble guanylyl cyclase (sGC)-cGMP signalling pathway appears to have far-reaching therapeutic potential. The discovery of sGC stimulators and activators marked a milestone in the field of NO-sGC-cGMP pharmacology, enabling NO-independent and long-acting enhancement of cGMP signalling without the formation of NO-derived radicals. Over a decade ago, the sGC stimulator riociguat was approved for the treatment of pulmonary arterial hypertension (PAH) and chronic thromboembolic PH (CTEPH). More recently, the sGC stimulator vericiguat was approved for symptomatic chronic HF. A number of sGC activators are currently being investigated for the treatment of chronic kidney diseases. This review summarizes the evidence for NO-sGC-cGMP signalling in the regulation of cardiovascular and cardiac function, focusing on preclinical and clinical evidence for sGC stimulators and sGC activators in HF subtypes. Promising results have been observed in clinical trials of HF with reduced ejection fraction (HFrEF),

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30104 - Pharmacology and pharmacy

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Biochemical pharmacology

  • ISSN

    0006-2952

  • e-ISSN

    1873-2968

  • Volume of the periodical

    242

  • Issue of the periodical within the volume

    December 2025 (Part 3)

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    17

  • Pages from-to

    "art.no. 117363"

  • UT code for WoS article

    001586015900003

  • EID of the result in the Scopus database

    2-s2.0-105020616435