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The essential transcription factor for granulocyte differentiation, CCAAT/enhancer binding protein alpha (CEBPA), and its mutations or inhibition associated with acute myeloid leukemias

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023736%3A_____%2F12%3A00010462" target="_blank" >RIV/00023736:_____/12:00010462 - isvavai.cz</a>

  • Result on the web

  • DOI - Digital Object Identifier

Alternative languages

  • Result language

    angličtina

  • Original language name

    The essential transcription factor for granulocyte differentiation, CCAAT/enhancer binding protein alpha (CEBPA), and its mutations or inhibition associated with acute myeloid leukemias

  • Original language description

    CEBP? induces myeloid differentiation via up-regulation of specific genes involved in granulocyte maturation and in inhibition of myeloid cell proliferation. CEBPA is intronless and generates two isoforms as a result of the differential utilization of alternate translation start codon. CEBP? p30 inhibits DNA binding of CEBP? p42 and its transactivation fiction in the expression of key genes in granulocytic differentiation. CEBPA mutation a methylation of its promoter were detected in acute myeloid leukemia and have prognostic significance.

  • Czech name

  • Czech description

Classification

  • Type

    C - Chapter in a specialist book

  • CEP classification

    FD - Oncology and haematology

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/LC06044" target="_blank" >LC06044: Center for Experimental Hematology</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>Z - Vyzkumny zamer (s odkazem do CEZ)

Others

  • Publication year

    2012

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Book/collection name

    Granulocytes: production, types and roles in disease

  • ISBN

    978-1-61942-806-5

  • Number of pages of the result

    36

  • Pages from-to

    33-68

  • Number of pages of the book

    142

  • Publisher name

    Nova Science Publishers

  • Place of publication

    New York

  • UT code for WoS chapter