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Attenuated cell cycle and DNA damage response transcriptome signatures and overrepresented cell adhesion processes imply accelerated progression in patients with lower‐risk myelodysplastic neoplasms

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023736%3A_____%2F24%3A00013596" target="_blank" >RIV/00023736:_____/24:00013596 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11110/24:10473670 RIV/00216208:11310/24:10473670 RIV/61989592:15110/24:73626530 RIV/00064165:_____/24:10473670

  • Result on the web

    <a href="https://doi.org/10.1002/ijc.34834" target="_blank" >https://doi.org/10.1002/ijc.34834</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/ijc.34834" target="_blank" >10.1002/ijc.34834</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Attenuated cell cycle and DNA damage response transcriptome signatures and overrepresented cell adhesion processes imply accelerated progression in patients with lower‐risk myelodysplastic neoplasms

  • Original language description

    Patients with myelodysplastic neoplasms (MDS) are classified according to the risk of acute myeloid leukemia transformation. Some lower-risk MDS patients (LR-MDS) progress rapidly despite expected good prognosis. Using diagnostic samples, we aimed to uncover the mechanisms of this accelerated progression at the transcriptome level. DNA damage response (DDR) and energy metabolism-related pathways were downregulated in prMDS samples, whereas cell adhesion processes were upregulated. Also, prMDS samples showed high levels of aberrant splicing and global lncRNA expression that may contribute to the attenuation of DDR pathways. The attenuation of DDR-related gene-expression signature may refine risk assessment in LR-MDS patients.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30205 - Hematology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2024

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    International journal of cancer

  • ISSN

    0020-7136

  • e-ISSN

  • Volume of the periodical

    154

  • Issue of the periodical within the volume

    9

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    17

  • Pages from-to

    1652-1668

  • UT code for WoS article

    001136704500001

  • EID of the result in the Scopus database

    2-s2.0-85181529959