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Hyperhomocysteinemia in Patients on Antiepileptic Drugs - Relation to the Mutation of the MTHFR Gene

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00023884%3A_____%2F01%3A00004845" target="_blank" >RIV/00023884:_____/01:00004845 - isvavai.cz</a>

  • Result on the web

  • DOI - Digital Object Identifier

Alternative languages

  • Result language

    angličtina

  • Original language name

    Hyperhomocysteinemia in Patients on Antiepileptic Drugs - Relation to the Mutation of the MTHFR Gene

  • Original language description

    The causes of hyperhomocysteinemia are multifactorial and include both genetic and acquired components (deficiency of vitamins, drug interference). Treatment with some antiepileptic drugs - phenytoin, carbamazepine, primidone or phenobarbital, (hepatic microsomal enzyme inductors) or their combinations is associated with a risk of increased plasma total homocysteine concentrations and deficiencies in vitamins important for homocysteine remethylation., ,

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    FP - Other medical fields

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/NE5489" target="_blank" >NE5489: Differentiation of mild hyperhomocysteinemia, its diagnostic significance and possible use as the selective screening in population of children with risk for ca</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2001

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Clinical Chemistry and Laboratory Medicine

  • ISSN

  • e-ISSN

  • Volume of the periodical

    39

  • Issue of the periodical within the volume

    suppl.

  • Country of publishing house

    DE - GERMANY

  • Number of pages

    1

  • Pages from-to

    "S341"

  • UT code for WoS article

  • EID of the result in the Scopus database