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The role of adipose tissue immune cells in obesity and low-grade inflammation

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00064165%3A_____%2F14%3A10288212" target="_blank" >RIV/00064165:_____/14:10288212 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11110/14:10288212

  • Result on the web

    <a href="http://dx.doi.org/10.1530/JOE-14-0283" target="_blank" >http://dx.doi.org/10.1530/JOE-14-0283</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1530/JOE-14-0283" target="_blank" >10.1530/JOE-14-0283</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    The role of adipose tissue immune cells in obesity and low-grade inflammation

  • Original language description

    Adipose tissue (AT) lies at the crossroad of nutrition, metabolism, and immunity; AT inflammation was proposed as a central mechanism connecting obesity with its metabolic and vascular complications. Resident immune cells constitute the second largest ATcellular component after adipocytes and as such play important roles in the maintenance of AT homeostasis. Obesity-induced changes in their number and activity result in the activation of local and later systemic inflammatory response, marking the transition from simple adiposity to diseases such as type 2 diabetes mellitus, arterial hypertension, and ischemic heart disease. This review has focused on the various subsets of immune cells in AT and their role in the development of AT inflammation and obesity-induced insulin resistance.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    FB - Endocrinology, diabetology, metabolism, nutrition

  • OECD FORD branch

Result continuities

  • Project

    <a href="/en/project/NT13299" target="_blank" >NT13299: Molecular mechanisms of subclinical inflammation in adipose tissue and its role in the etiopathogenesis of type 2 diabetes mellitus</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)<br>I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2014

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Endocrinology

  • ISSN

    0022-0795

  • e-ISSN

  • Volume of the periodical

    222

  • Issue of the periodical within the volume

    3

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    15

  • Pages from-to

    "R113"-"R127"

  • UT code for WoS article

    000341881100003

  • EID of the result in the Scopus database