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High hydrostatic pressure induces immunogenic cell death in human tumor cells

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00064203%3A_____%2F14%3A10292981" target="_blank" >RIV/00064203:_____/14:10292981 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11130/14:10292981

  • Result on the web

    <a href="http://dx.doi.org/10.1002/ijc.28766" target="_blank" >http://dx.doi.org/10.1002/ijc.28766</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1002/ijc.28766" target="_blank" >10.1002/ijc.28766</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    High hydrostatic pressure induces immunogenic cell death in human tumor cells

  • Original language description

    Recent studies have identified molecular events characteristic of immunogenic cell death (ICD), including surface exposure of calreticulin (CRT), the heat shock proteins HSP70 and HSP90, the release of high-mobility group box protein 1 (HMGB1) and the release of ATP from dying cells. We investigated the potential of high hydrostatic pressure (HHP) to induce ICD in human tumor cells. HHP induced the rapid expression of HSP70, HSP90 and CRT on the cell surface. HHP also induced the release of HMGB1 and ATP. The interaction of dendritic cells (DCs) with HHP-treated tumor cells led to a more rapid rate of DC phagocytosis, upregulation of CD83, CD86 and HLA-DR and the release of interleukin IL-6, IL-12p70 and TNF-alpha. DCs pulsed with tumor cells killed byHHP induced high numbers of tumor-specific T cells. DCs pulsed with HHP-treated tumor cells also induced the lowest number of regulatory T cells. In addition, we found that the key features of the endoplasmic reticulum stress-mediated ap

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    FD - Oncology and haematology

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2014

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    International Journal of Cancer

  • ISSN

    0020-7136

  • e-ISSN

  • Volume of the periodical

    135

  • Issue of the periodical within the volume

    5

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    13

  • Pages from-to

    1165-1177

  • UT code for WoS article

    000337608300017

  • EID of the result in the Scopus database