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Schizophrenia risk from complex variation of complement component 4

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00064203%3A_____%2F16%3A10337226" target="_blank" >RIV/00064203:_____/16:10337226 - isvavai.cz</a>

  • Alternative codes found

    RIV/00216208:11130/16:10337226

  • Result on the web

    <a href="http://dx.doi.org/10.1038/nature16549" target="_blank" >http://dx.doi.org/10.1038/nature16549</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1038/nature16549" target="_blank" >10.1038/nature16549</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Schizophrenia risk from complex variation of complement component 4

  • Original language description

    Schizophrenia is a heritable brain illness with unknown pathogenic mechanisms. Schizophrenia's strongest genetic association at a population level involves variation in the major histocompatibility complex (MHC) locus, but the genes and molecular mechanisms accounting for this have been challenging to identify. Here we show that this association arises in part from many structurally diverse alleles of the complement component 4 (C4) genes. We found that these alleles generated widely varying levels of C4A and C4B expression in the brain, with each common C4 allele associating with schizophrenia in proportion to its tendency to generate greater expression of C4A. Human C4 protein localized to neuronal synapses, dendrites, axons, and cell bodies. In mice, C4 mediated synapse elimination during postnatal development. These results implicate excessive complement activity in the development of schizophrenia and may help explain the reduced numbers of synapses in the brains of individuals with schizophrenia.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

  • Continuities

    I - Institucionalni podpora na dlouhodoby koncepcni rozvoj vyzkumne organizace

Others

  • Publication year

    2016

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Nature

  • ISSN

    0028-0836

  • e-ISSN

  • Volume of the periodical

    530

  • Issue of the periodical within the volume

    7589

  • Country of publishing house

    GB - UNITED KINGDOM

  • Number of pages

    7

  • Pages from-to

    177-183

  • UT code for WoS article

    000369916700030

  • EID of the result in the Scopus database

    2-s2.0-84958074030