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Myeloperoxidase induces the priming of platelets

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00159816%3A_____%2F13%3A00060645" target="_blank" >RIV/00159816:_____/13:00060645 - isvavai.cz</a>

  • Alternative codes found

    RIV/68081707:_____/13:00394796 RIV/00216224:14310/13:00081894

  • Result on the web

    <a href="http://dx.doi.org/10.1016/j.freeradbiomed.2013.04.014" target="_blank" >http://dx.doi.org/10.1016/j.freeradbiomed.2013.04.014</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.freeradbiomed.2013.04.014" target="_blank" >10.1016/j.freeradbiomed.2013.04.014</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Myeloperoxidase induces the priming of platelets

  • Original language description

    The release of myeloperoxidase (MPO) from polymorphonuclear neutrophils is a hallmark of vascular inflammation and contributes to the pathogenesis of vascular inflammatory processes. However, the effects of MPO on platelets as a contributory mechanism invascular inflammatory diseases remain unknown. Thus, MPO interaction with platelets and its effects on platelet function were examined. First, dose-dependent binding of MPO (between 1.7 and 13.8 nM) to both human and mouse platelets was observed. This was in direct contrast to the absence of MPO in megakaryocytes. MPO was localized both on the surface of and inside platelets. Cytoskeleton inhibition did not prevent MPO localization inside the three-dimensional platelet structure. MPO peroxidase activity was preserved upon the MPO binding to platelets. MPO sequestered in platelets catabolized NO, documented by the decreased production of NO (on average, an approximately 2-fold decrease). MPO treatment did not affect the viability of pla

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    CE - Biochemistry

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2013

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Free radical biology and medicine

  • ISSN

    0891-5849

  • e-ISSN

  • Volume of the periodical

    61

  • Issue of the periodical within the volume

    April

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    13

  • Pages from-to

    357-369

  • UT code for WoS article

    000320687300035

  • EID of the result in the Scopus database