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Instability restricts signaling of multiple fibroblast growth factors

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00159816%3A_____%2F15%3A00062877" target="_blank" >RIV/00159816:_____/15:00062877 - isvavai.cz</a>

  • Alternative codes found

    RIV/68081707:_____/15:00445257 RIV/67985904:_____/15:00445257 RIV/00216224:14110/15:00080926 RIV/62157124:16170/15:43873501

  • Result on the web

    <a href="http://dx.doi.org/10.1007/s00018-015-1856-8" target="_blank" >http://dx.doi.org/10.1007/s00018-015-1856-8</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1007/s00018-015-1856-8" target="_blank" >10.1007/s00018-015-1856-8</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Instability restricts signaling of multiple fibroblast growth factors

  • Original language description

    Fibroblast growth factors (FGFs) deliver extracellular signals that govern many developmental and regenerative processes, but the mechanisms regulating FGF signaling remain incompletely understood. Here, we explored the relationship between intrinsic stability of FGF proteins and their biological activity for all 18 members of the FGF family. We report that FGF1, FGF3, FGF4, FGF6, FGF8, FGF9, FGF10, FGF16, FGF17, FGF18, FGF20, and FGF22 exist as unstable proteins, which are rapidly degraded in cell cultivation media. Biological activity of FGF1, FGF3, FGF4, FGF6, FGF8, FGF10, FGF16, FGF17, and FGF20 is limited by their instability, manifesting as failure to activate FGF receptor signal transduction over long periods of time, and influence specific cell behavior in vitro and in vivo. Stabilization via exogenous heparin binding, introduction of stabilizing mutations or lowering the cell cultivation temperature rescues signaling of unstable FGFs. Thus, the intrinsic ligand instability is an important elementary level of regulation in the FGF signaling system.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>x</sub> - Unclassified - Peer-reviewed scientific article (Jimp, Jsc and Jost)

  • CEP classification

    EB - Genetics and molecular biology

  • OECD FORD branch

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2015

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Cellular and Molecular Life Sciences

  • ISSN

    1420-682X

  • e-ISSN

  • Volume of the periodical

    72

  • Issue of the periodical within the volume

    12

  • Country of publishing house

    CH - SWITZERLAND

  • Number of pages

    15

  • Pages from-to

    2445-2459

  • UT code for WoS article

    000354883500014

  • EID of the result in the Scopus database