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Vascular Invasion of the Dental Epithelium Is Essential for Ameloblasts

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11110%2F25%3A10500008" target="_blank" >RIV/00216208:11110/25:10500008 - isvavai.cz</a>

  • Result on the web

    <a href="https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=YXfsP54sXC" target="_blank" >https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=YXfsP54sXC</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1177/00220345251341850" target="_blank" >10.1177/00220345251341850</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Vascular Invasion of the Dental Epithelium Is Essential for Ameloblasts

  • Original language description

    The tooth is a highly vascularized organ. During odontogenesis, blood vessels enter the forming tooth through the dental papilla and surround the dental epithelium from the cap stage. We show that during the late bell stage, endothelial cells invade the outer enamel epithelium (OEE) and migrate through the stellate reticulum to vascularize the forming ameloblast layer. This migration was evident in both mouse and human tooth germs and is likely to represent a conserved mechanism. Migration was coordinated by dynamic changes in Vegf expression in the dental epithelium, with expression at the OEE at the bell stage shifting toward the ameloblast layer. Invasion through the OEE involved loss of integrity of the basement membrane, downregulation of tight junctions, and apoptosis of some OEE cells. Changes in the OEE were dependent on Hedgehog signaling, with a failure to invade in K14creSmoothenedfl/fl mice, where epithelial cells cannot respond to Hedgehog signaling. The impact of failed migration through the OEE was followed in Cdh5creERT2 Vegfr2fl/fl mutant mice, where the endothelial cells cannot respond to vascular endothelial growth factor (VEGF). Failure of OEE invasion resulted in differentiation defects and extensive cell death of the ameloblast layer, highlighting the essential requirement for vascularization for development of this layer. Our results reveal the essential role of Hedgehog and VEGF signaling in correct vascularization of the tooth germ epithelial layers, allowing breakdown of the OEE and targeting endothelial cell migration into the epithelium. A better understanding of the molecular regulation of endothelial cells will help decipher how this cell population interacts with different cells of the enamel organ and will aid in attempts to revascularize teeth.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30106 - Anatomy and morphology (plant science to be 1.6)

Result continuities

  • Project

    <a href="/en/project/GA23-06660S" target="_blank" >GA23-06660S: Physiological dynamics of odontogenesis in context of surrounding structures</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Journal of Dental Research

  • ISSN

    0022-0345

  • e-ISSN

    1544-0591

  • Volume of the periodical

    104

  • Issue of the periodical within the volume

    13

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    9

  • Pages from-to

    1547-1555

  • UT code for WoS article

    001517996200001

  • EID of the result in the Scopus database

    2-s2.0-105010077692