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Control of myeloid lineage fidelity and response to stimuli by ISWI-enforced nucleosome phasing

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11110%2F25%3A10504949" target="_blank" >RIV/00216208:11110/25:10504949 - isvavai.cz</a>

  • Result on the web

    <a href="https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=36czEYKxN~" target="_blank" >https://verso.is.cuni.cz/pub/verso.fpl?fname=obd_publikace_handle&handle=36czEYKxN~</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.immuni.2025.09.002" target="_blank" >10.1016/j.immuni.2025.09.002</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Control of myeloid lineage fidelity and response to stimuli by ISWI-enforced nucleosome phasing

  • Original language description

    The interplay between chromatin remodelers and pioneer transcription factors (TFs) regulates cis-regulatory element accessibility to maintain cell identity and transcriptional fidelity. We investigated the impact of imitatiation and activation, focusing on SMARCA5, the sole ISWI ATPase in myeloid cells. Conditional Smarca5 a pioneer TF essential for myeloid identity, without altering PU.1 occupancy. However, SMARCA5 loss increased accessibility at motifs bound by C/EBP beta, a weak pioneer TF, enabling binding to regulatory regions active in non-hematopoietic lineages and causing lineage-inappropriate transcription. These changes also increased accessibility at sites bound by stimulus-induced TFs, leading to macrophage hyperactivation and mis-expression of stimulus-inappropriate genes. Thus, SMARCA5-dependent nucleosome phasing respecification and activation, with likely similar roles in other immune cell types.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30205 - Hematology

Result continuities

  • Project

    Result was created during the realization of more than one project. More information in the Projects tab.

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2025

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Immunity

  • ISSN

    1074-7613

  • e-ISSN

    1097-4180

  • Volume of the periodical

    58

  • Issue of the periodical within the volume

    10

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    26

  • Pages from-to

    2402-"2418.e8"

  • UT code for WoS article

    001600265600003

  • EID of the result in the Scopus database

    2-s2.0-105018211900