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Diabetes in stiff-person syndrome

The result's identifiers

  • Result code in IS VaVaI

    <a href="https://www.isvavai.cz/riv?ss=detail&h=RIV%2F00216208%3A11120%2F23%3A43925955" target="_blank" >RIV/00216208:11120/23:43925955 - isvavai.cz</a>

  • Result on the web

    <a href="https://doi.org/10.1016/j.tem.2023.07.005" target="_blank" >https://doi.org/10.1016/j.tem.2023.07.005</a>

  • DOI - Digital Object Identifier

    <a href="http://dx.doi.org/10.1016/j.tem.2023.07.005" target="_blank" >10.1016/j.tem.2023.07.005</a>

Alternative languages

  • Result language

    angličtina

  • Original language name

    Diabetes in stiff-person syndrome

  • Original language description

    Anti-glutamic acid decarboxylase (GAD) autoantibodies are a hallmark of stiff-person syndrome (SPS) and insulin-dependent diabetes mellitus (IDDM). However, patients with concurrent IDDM and SPS often manifest insulin resistance, and SPS-associated IDDM probably has heterogeneous causes. Some patients manifest IDDM associated only with high titers of anti-GAD65 caused by SPS. By contrast, other patients develop IDDM only after being treated with high-dose corticosteroids or they progress to insulin dependency following their treatment with high-dose corticosteroids. The profile of autoantibodies differs markedly between type 1 diabetes mellitus (T1DM), late-onset diabetes mellitus, and SPS-associated IDDM. Therefore, as with new-onset diabetes after transplantation (NODAT), SPS-associated IDDM should be classified as a specific diabetes entity, the pathophysiology of which requires increased attention.

  • Czech name

  • Czech description

Classification

  • Type

    J<sub>imp</sub> - Article in a specialist periodical, which is included in the Web of Science database

  • CEP classification

  • OECD FORD branch

    30202 - Endocrinology and metabolism (including diabetes, hormones)

Result continuities

  • Project

    <a href="/en/project/NU23-06-00045" target="_blank" >NU23-06-00045: Development of an in vitro model for the study of renal dysfunction due to pharmacotherapy</a><br>

  • Continuities

    P - Projekt vyzkumu a vyvoje financovany z verejnych zdroju (s odkazem do CEP)

Others

  • Publication year

    2023

  • Confidentiality

    S - Úplné a pravdivé údaje o projektu nepodléhají ochraně podle zvláštních právních předpisů

Data specific for result type

  • Name of the periodical

    Trends in Endocrinology & Metabolism

  • ISSN

    1043-2760

  • e-ISSN

    1879-3061

  • Volume of the periodical

    34

  • Issue of the periodical within the volume

    10

  • Country of publishing house

    US - UNITED STATES

  • Number of pages

    12

  • Pages from-to

    640-651

  • UT code for WoS article

    001079253900001

  • EID of the result in the Scopus database

    2-s2.0-85168336494